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8th November 2018 18:43
Link to Post #21
Re: Tinnitus. sigh. Anyone here experience this?
I've pulled up a lot of research on this to look for the commonalities behind the nerve inflammation, the result is the inflammation is coming from endotoxins.
Gram-negative bacteria release dangerous endotoxins primarily when they are killed.
This can occur when the immune system or antibiotics attack the bacteria and break down their cell walls.
When the cell walls are broken, fragments of the outer membrane are released into the body, including the endotoxins.
Tinnitus will often occur in one ear, appear to get stronger and quieter and are modulated by stress. One can mimic the tinnutis sound by tensing the jaw muscles like one is going to grimace, squinting.. example:
Increasing pressure on the facial and jaw nerves and those coming from the teeth causes triggering, and the "sound" appears.
Notable gram negative bacteria are Escherichia coli, Salmonella, Shigella, Pseudomonas, Neisseria, Haemophilus influenzae, Bordetella pertussis and Vibrio cholerae.
One member mentions having taken an antibiotic and there is a subsequent development of tinnitus.. Logical especially with the gram-negative bacteria dying off, the toxins released from the dying BACTERIA (not the antibiotic) were endotoxic. All of those bacteria can exist in the mouth, nasal, sinus, gut. When those bacteria are killed naturally by the body's immune system, or die off from a different pro-biotic species being introduced, or from antibiotics, we all run the risk of a massive dose of endotoxic poisons entering our blood stream.
So any solutions to treating tinnitus (and other nerve inflammations, chronic pain, etc.) are hopeful methods if they can address the hypersensitivity of the nerves..
What specifically is being triggered by the toxin? Where does the toxin come from in the gram-negative bacteria? From the Lipopolysaccharides (LPS) or the outer "skin" of the bacteria.
The toxin triggers in the body, sepsis, a type of inflammation, where the cells leak (and put pressure on the nerves), and create a strong pain. A lower level of "pain" from the swelling in the nerves can evoke the tinnitus symptoms.
Anything then activating a nerve sensitized by endotoxins can re-trigger the tinnitus symptoms.. Salicylate, the active component of aspirin, is known to induce tinnitus.
There was mention of using curcumin (post 12 above), which will also reduce inflammation.. But the cause comes back to the endotoxin hypersensitization. That came from the gut, from the bacterial dying off, from bacterial "flu", all of which create different levels of that form of "toxic shock".
Other than tinnitus one can have gut symptoms, such as "tender gut" a feeling of persistent malaise (a general sense of something is wrong but i can't put my finger on it), low level body pain, symptoms of bloating, periodic chills.. the list goes on and on, some folks have called it the Fibromyalgia syndrome. That fibromyalsia issue affects the muscles and soft tissue. Symptoms include chronic muscle pain, fatigue, sleep problems, and painful tender points or trigger points, which can be relieved through medications, lifestyle changes and stress management.
But getting to the cause, the gram-bacterial die-off triggers the "tumor-necrosis-factor-alpha" within the cell's gene's, which triggers the inflammation which triggers pain receptors which triggers "flooding", and so forth, a nasty cascade of symptoms which left unchecked can lead to "toxic shock".
Then getting into the nitty-gritty, to stop that inflammation cycle one needs to reach it at the target sites..
A study of using NALTREXONE, in 50 milligram dosing levels stops tinnitus by directly plugging up the opiod receptors.
Naltrexone is used by emergency personnel for instance to treat a morphine or heroin overdose. Opiod overdose can induce endotoxic shock. Naltrexone, an opioid antagonist, is usually prescribed for opioid and alcohol addiction.
One can pull up this article: https://www.dailymedicaldiscoveries....-ears-forever/ to see how they researched the tinnitus and found the endotoxin link.
In seeing the nerve receptors triggered (with the inflammation reaction resultant), if one takes morphine codeine or heroin, one can exacerbate (make worse) cellular leakage. Severe cellular leakage does exist in SEPSIS (see my thread on that in the Forum, Alt Medicine).. And that severe cellular leakage will not just cause ringing in the ears, it can cause a lot of pain and eventual death from organ failure.
In other words, the nerve inflammation has to be reduced without using morphines or morphine-like substances (and remember the body produces it's own natural morphines).. so to properly "modulate" (vary the intensity level as needed intelligently) the body's endorphins is important.
Turn on pain, endorphins increase, tinnitus goes up, turn off pain by reducing inflammation and the tinnitus drops and the overall body malaise sensation corrects..
reference study:
Last edited by Bob; 8th November 2018 at 19:04.
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8th November 2018 19:04
Link to Post #22