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View Full Version : Statin drugs kill one cell at a time! Plz share this information -It will save lives!



Kimberley
4th May 2012, 18:11
IMPORTANT-How Statin Drugs REALLY Lower Cholesterol
(And Kill You One Cell at a Time) Please share this!!

25% of the population of 1st world countries take statin drugs!!


Here is my interview on youtube with Dr Hannah Yoseph and James Yoseph recorded April 25, 2011

Thank you for sharing this with others...this information WILL save lives and open eyes!!!


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"How Statin Drugs REALLY Lower Cholesterol (And Kill You One Cell at a Time)" web sites.

http://statininjuryvictims.blogspot.com/ http://statinsideeffects.webs.com/


The Yosephs have written a stunning exposé. In simple language they reveal the science, the corruption and the enormous conspiracy it took to bring statins to market. They report the research, the fraud and the facts like a detective in hot pursuit of a Nazi war criminal. Once picked up it cannot be put down until the reading is done. It is riveting. They have accomplished the impossible: they have made both complex science and medical history fascinating to read. What could not be done in an exposé they accomplished with almost unbelievable ease. It will change your paradigms about medicine forever. Read it!



Here is a direct link to my internet radio show "Living In The NOW" that features this same interview

http://www.blogtalkradio.com/belovenow/2012/05/01/livinginthenowimportant-how-statin-drugs-kill-you

You can download the audio to any computer and audio player by clicking on the little blue letters "Download this episode" under the photo screen.

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You are invited to listen to "Living In The NOW!! A weekly show on Tuesday Live at Noon EST and archived.

http://www.blogtalkradio.com/belovenow

There are over 3 years of archives available with hundreds of conscious thought leaders such as Gregg Braden, Bruce Lipton, Dr Brian Weiss, Jim Marrs, Anita Moorjani, Santos Bonacci, George Kavassilias, Dr Carl Calleman, and many many more...

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You cay also be interested in this short video about the book
"How Statin Drugs REALLY Lower Cholesterol (And Kill You One Cell at a Time)"
robzC_tZZ1o

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mexrph
4th May 2012, 18:38
Statin drugs are POISON. I am a retired pharmacist and have spent 400+ hours on the internet investigating this. Statin drugs are part of the NWO plan to depopulate the earth. DO NOT TAKE STATIN DRUGS AND GET ALL FAMILY MEMBERS OFF THEM. Also, DO NOT TAKE ANY CHOLESTEROL LOWERING DRUG. Read Dr Uffe Ravnskov's brilliant article: "The Benefits of High Cholesterol". It will blow your mind if you have been brainwashed to think that high cholesterol is bad.

Eligos
4th May 2012, 18:42
I was told by a Dr. 3 years ago (a walk-in clinic) that my Cholesterol was on the high side and that I should get started immediately on Statins. I looked at her, smiled and said, "I will NEVER take statins". She looked at me puzzled and said "well that is your choice of course". I left and never saw her again. I already knew a lot about how bad they were and I could tell she wasn't in the mood to be schooled by me. What's the point anyway, they believe their crap.

nearing
4th May 2012, 18:44
Dr Uffe Ravnskov's brilliant article: "The Benefits of High Cholesterol" (http://www.westonaprice.org/cardiovascular-disease/benefits-of-high-cholesterol)

People with high cholesterol live the longest. This statement seems so incredible that it takes a long time to clear one´s brainwashed mind to fully understand its importance. Yet the fact that people with high cholesterol live the longest emerges clearly from many scientific papers. Consider the finding of Dr. Harlan Krumholz of the Department of Cardiovascular Medicine at Yale University, who reported in 1994 that old people with low cholesterol died twice as often from a heart attack as did old people with a high cholesterol.1 Supporters of the cholesterol campaign consistently ignore his observation, or consider it as a rare exception, produced by chance among a huge number of studies finding the opposite.
But it is not an exception; there are now a large number of findings that contradict the lipid hypothesis. To be more specific, most studies of old people have shown that high cholesterol is not a risk factor for coronary heart disease. This was the result of my search in the Medline database for studies addressing that question.2 Eleven studies of old people came up with that result, and a further seven studies found that high cholesterol did not predict all-cause mortality either.
Now consider that more than 90 % of all cardiovascular disease is seen in people above age 60 also and that almost all studies have found that high cholesterol is not a risk factor for women.2 This means that high cholesterol is only a risk factor for less than 5 % of those who die from a heart attack.
But there is more comfort for those who have high cholesterol; six of the studies found that total mortality was inversely associated with either total or LDL-cholesterol, or both. This means that it is actually much better to have high than to have low cholesterol if you want to live to be very old.
High Cholesterol Protects Against Infection

Many studies have found that low cholesterol is in certain respects worse than high cholesterol. For instance, in 19 large studies of more than 68,000 deaths, reviewed by Professor David R. Jacobs and his co-workers from the Division of Epidemiology at the University of Minnesota, low cholesterol predicted an increased risk of dying from gastrointestinal and respiratory diseases.3
Most gastrointestinal and respiratory diseases have an infectious origin. Therefore, a relevant question is whether it is the infection that lowers cholesterol or the low cholesterol that predisposes to infection? To answer this question Professor Jacobs and his group, together with Dr. Carlos Iribarren, followed more than 100,000 healthy individuals in the San Francisco area for fifteen years. At the end of the study those who had low cholesterol at the start of the study had more often been admitted to the hospital because of an infectious disease.4,5 This finding cannot be explained away with the argument that the infection had caused cholesterol to go down, because how could low cholesterol, recorded when these people were without any evidence of infection, be caused by a disease they had not yet encountered? Isn´t it more likely that low cholesterol in some way made them more vulnerable to infection, or that high cholesterol protected those who did not become infected? Much evidence exists to support that interpretation.
Low Cholesterol and HIV/AIDS

Young, unmarried men with a previous sexually transmitted disease or liver disease run a much greater risk of becoming infected with HIV virus than other people. The Minnesota researchers, now led by Dr. Ami Claxton, followed such individuals for 7-8 years. After having excluded those who became HIV-positive during the first four years, they ended up with a group of 2446 men. At the end of the study, 140 of these people tested positive for HIV; those who had low cholesterol at the beginning of the study were twice as likely to test postitive for HIV compared with those with the highest cholesterol.6
Similar results come from a study of the MRFIT screenees, including more than 300,000 young and middle-aged men, which found that 16 years after the first cholesterol analysis the number of men whose cholesterol was lower than 160 and who had died from AIDS was four times higher than the number of men who had died from AIDS with a cholesterol above 240.7
Cholesterol and Chronic Heart Failure

Heart disease may lead to a weakening of the heart muscle. A weak heart means that less blood and therefore less oxygen is delivered to the arteries. To compensate for the decreased power, the heart beat goes up, but in severe heart failure this is not sufficient. Patients with severe heart failure become short of breath because too little oxygen is delivered to the tissues, the pressure in their veins increases because the heart cannot deliver the blood away from the heart with sufficient power, and they become edematous, meaning that fluid accumulates in the legs and in serious cases also in the lungs and other parts of the body. This condition is called congestive or chronic heart failure.
There are many indications that bacteria or other microorganisms play an important role in chronic heart failure. For instance, patients with severe chronic heart failure have high levels of endotoxin and various types of cytokines in their blood. Endotoxin, also named lipopolysaccharide, is the most toxic substance produced by Gram-negative bacteria such as Escherichia coli, Klebsiella, Salmonella, Serratia and Pseudomonas. Cytokines are hormones secreted by white blood cells in their battle with microorganisms; high levels of cytokines in the blood indicate that inflammatory processes are going on somewhere in the body.
The role of infections in chronic heart failure has been studied by Dr. Mathias Rauchhaus and his team at the Medical Department, Martin-Luther-University in Halle, Germany (Universitätsklinik und Poliklinik für Innere Medizin III, Martin-Luther-Universität, Halle). They found that the strongest predictor of death for patients with chronic heart failure was the concentration of cytokines in the blood, in particular in patients with heart failure due to coronary heart disease.8 To explain their finding they suggested that bacteria from the gut may more easily penetrate into the tissues when the pressure in the abdominal veins is increased because of heart failure. In accordance with this theory, they found more endotoxin in the blood of patients with congestive heart failure and edema than in patients with non-congestive heart failure without edema, and endotoxin concentrations decreased significantly when the heart’s function was improved by medical treatment.9
A simple way to test the functional state of the immune system is to inject antigens from microorganisms that most people have been exposed to, under the skin. If the immune system is normal, an induration (hard spot) will appear about 48 hours later at the place of the injection. If the induration is very small, with a diameter of less than a few millimeters, this indicates the presence of "anergy," a reduction in or failure of response to recognize antigens. In accordance, anergy has been found associated with an increased risk of infection and mortality in healthy elderly individuals, in surgical patients and in heart transplant patients.10
Dr. Donna Vredevoe and her group from the School of Nursery and the School of Medicine, University of California at Los Angeles tested more than 200 patients with severe heart failure with five different antigens and followed them for twelve months. The cause of heart failure was coronary heart disease in half of them and other types of heart disease (such as congenital or infectious valvular heart disease, various cardiomyopathies and endocarditis) in the rest. Almost half of all the patients were anergic, and those who were anergic and had coronary heart disease had a much higher mortality than the rest.10
Now to the salient point: to their surprise the researchers found that mortality was higher, not only in the patients with anergy, but also in the patients with the lowest lipid values, including total cholesterol, LDL-cholesterol and HDL-cholesterol as well as triglycerides.
The latter finding was confirmed by Dr. Rauchhaus, this time in co-operation with researchers at several German and British university hospitals. They found that the risk of dying for patients with chronic heart failure was strongly and inversely associated with total cholesterol, LDL-cholesterol and also triglycerides; those with high lipid values lived much longer than those with low values.11,12
Other researchers have made similar observations. The largest study has been performed by Professor Gregg C. Fonorow and his team at the UCLA Department of Medicine and Cardiomyopathy Center in Los Angeles.13 The study, led by Dr. Tamara Horwich, included more than a thousand patients with severe heart failure. After five years 62 percent of the patients with cholesterol below 129 mg/l had died, but only half as many of the patients with cholesterol above 223 mg/l.
When proponents of the cholesterol hypothesis are confronted with findings showing a bad outcome associated with low cholesterol--and there are many such observations--they usually argue that severely ill patients are often malnourished, and malnourishment is therefore said to cause low cholesterol. However, the mortality of the patients in this study was independent of their degree of nourishment; low cholesterol predicted early mortality whether the patients were malnourished or not.
Smith-Lemli-Opitz Syndrome

As discussed in The Cholesterol Myths (see sidebar), much evidence supports the theory that people born with very high cholesterol, so-called familial hypercholesterolemia, are protected against infection. But if inborn high cholesterol protects against infections, inborn low cholesterol should have the opposite effect. Indeed, this seems to be true.
Children with the Smith-Lemli-Opitz syndrome have very low cholesterol because the enzyme that is necessary for the last step in the body’s synthesis of cholesterol does not function properly. Most children with this syndrome are either stillborn or they die early because of serious malformations of the central nervous system. Those who survive are imbecile, they have extremely low cholesterol and suffer from frequent and severe infections. However, if their diet is supplemented with pure cholesterol or extra eggs, their cholesterol goes up and their bouts of infection become less serious and less frequent.14
Laboratory Evidence

Laboratory studies are crucial for learning more about the mechanisms by which the lipids exert their protective function. One of the first to study this phenomenon was Dr Sucharit Bhakdi from the Institute of Medical Microbiology, University of Giessen (Institut für Medizinsche Mikrobiologie, Justus-Liebig-Universität Gießen), Germany along with his team of researchers from various institutions in Germany and Denmark.15
Staphylococcus aureus α-toxin is the most toxic substance produced by strains of the disease-promoting bacteria called staphylococci. It is able to destroy a wide variety of human cells, including red blood cells. For instance, if minute amounts of the toxin are added to a test tube with red blood cells dissolved in 0.9 percent saline, the blood is hemolyzed, that is the membranes of the red blood cells burst and hemoglobin from the interior of the red blood cells leaks out into the solvent. Dr. Bhakdi and his team mixed purified α-toxin with human serum (the fluid in which the blood cells reside) and saw that 90 percent of its hemolyzing effect disappeared. By various complicated methods they identified the protective substance as LDL, the carrier of the so-called bad cholesterol. In accordance, no hemolysis occurred when they mixed α-toxin with purified human LDL, whereas HDL or other plasma constituents were ineffective in this respect.
Dr. Willy Flegel and his co-workers at the Department of Transfusion Medicine, University of Ulm, and the Institute of Immunology and Genetics at the German Cancer Research Center in Heidelberg, Germany (DRK-Blutspendezentrale und Abteilung für Transfusionsmedizin, Universität Ulm, und Deutsches Krebsforschungszentrum, Heidelberg) studied endotoxin in another way.16 As mentioned, one of the effects of endotoxin is that white blood cells are stimulated to produce cytokines. The German researchers found that the cytokine-stimulating effect of endotoxin on the white blood cells disappeared almost completely if the endotoxin was mixed with human serum for 24 hours before they added the white blood cells to the test tubes. In a subsequent study17 they found that purified LDL from patients with familial hypercholesterolemia had the same inhibitory effect as the serum.
LDL may not only bind and inactivate dangerous bacterial toxins; it seems to have a direct beneficial influence on the immune system also, possibly explaining the observed relationship between low cholesterol and various chronic diseases. This was the starting point for a study by Professor Matthew Muldoon and his team at the University of Pittsburgh, Pennsylvania. They studied healthy young and middle-aged men and found that the total number of white blood cells and the number of various types of white blood cells were significantly lower in the men with LDL-cholesterol below 160 mg/dl (mean 88.3 mg/l),than in men with LDL-cholesterol above 160 mg/l (mean 185.5 mg/l).18 The researchers cautiously concluded that there were immune system differences between men with low and high cholesterol, but that it was too early to state whether these differences had any importance for human health. Now, seven years later with many of the results discussed here, we are allowed to state that the immune-supporting properties of LDL-cholesterol do indeed play an important role in human health.
Animal Experiments

The immune systems in various mammals including human beings have many similarities. Therefore, it is interesting to see what experiments with rats and mice can tell us. Professor Kenneth Feingold at the Department of Medicine, University of California, San Francisco, and his group have published several interesting results from such research. In one of them they lowered LDL-cholesterol in rats by giving them either a drug that prevents the liver from secreting lipoproteins, or a drug that increases their disappearance. In both models, injection of endotoxin was followed by a much higher mortality in the low-cholesterol rats compared with normal rats. The high mortality was not due to the drugs because, if the drug-treated animals were injected with lipoproteins just before the injection of endotoxin, their mortality was reduced to normal.19
Dr. Mihai Netea and his team from the Departments of Internal and Nuclear Medicine at the University Hospital in Nijmegen, The Netherlands, injected purified endotoxin into normal mice, and into mice with familial hypercholesterolemia that had LDL-cholesterol four times higher than normal. Whereas all normal mice died, they had to inject eight times as much endotoxin to kill the mice with familial hypercholesterolemia. In another experiment they injected live bacteria and found that twice as many mice with familial hypercholesterolemia survived compared with normal mice.20
Other Protecting Lipids

As seen from the above, many of the roles played by LDL-cholesterol are shared by HDL. This should not be too surprising considering that high HDL-cholesterol is associated with cardiovascular health and longevity. But there is more.
Triglycerides, molecules consisting of three fatty acids linked to glycerol, are insoluble in water and are therefore carried through the blood inside lipoproteins, just as cholesterol. All lipoproteins carry triglycerides, but most of them are carried by a lipoprotein named VLDL (very low-density lipoprotein) and by chylomicrons, a mixture of emulsified triglycerides appearing in large amounts after a fat-rich meal, particularly in the blood that flows from the gut to the liver.
For many years it has been known that sepsis, a life-threatening condition caused by bacterial growth in the blood, is associated with a high level of triglycerides. The serious symptoms of sepsis are due to endotoxin, most often produced by gut bacteria. In a number of studies, Professor Hobart W. Harris at the Surgical Research Laboratory at San Francisco General Hospital and his team found that solutions rich in triglycerides but with practically no cholesterol were able to protect experimental animals from the toxic effects of endotoxin and they concluded that the high level of triglycerides seen in sepsis is a normal immune response to infection.21 Usually the bacteria responsible for sepsis come from the gut. It is therefore fortunate that the blood draining the gut is especially rich in triglycerides.
Exceptions

So far, animal experiments have confirmed the hypothesis that high cholesterol protects against infection, at least against infections caused by bacteria. In a similar experiment using injections of Candida albicans, a common fungus, Dr. Netea and his team found that mice with familial hypercholesterolemia died more easily than normal mice.22 Serious infections caused by Candida albicans are rare in normal human beings; however, they are mainly seen in patients treated with immunosuppressive drugs, but the finding shows that we need more knowledge in this area. However, the many findings mentioned above indicate that the protective effects of the blood lipids against infections in human beings seem to be greater than any possible adverse effects.
Cholesterol as a Risk Factor

Most studies of young and middle-aged men have found high cholesterol to be a risk factor for coronary heart disease, seemingly a contradiction to the idea that high cholesterol is protective. Why is high cholesterol a risk factor in young and middle-aged men? A likely explanation is that men of that age are often in the midst of their professional career. High cholesterol may therefore reflect mental stress, a well-known cause of high cholesterol and also a risk factor for heart disease. Again, high cholesterol is not necessarily the direct cause but may only be a marker. High cholesterol in young and middle-aged men could, for instance, reflect the body’s need for more cholesterol because cholesterol is the building material of many stress hormones. Any possible protective effect of high cholesterol may therefore be counteracted by the negative influence of a stressful life on the vascular system.
Response to Injury

In 1976 one of the most promising theories about the cause of atherosclerosis was the Response-to-Injury Hypothesis, presented by Russell Ross, a professor of pathology, and John Glomset, a professor of biochemistry and medicine at the Medical School, University of Washington in Seattle.23,24 They suggested that atherosclerosis is the consequence of an inflammatory process, where the first step is a localized injury to the thin layer of cells lining the inside of the arteries, the intima. The injury causes inflammation and the raised plaques that form are simply healing lesions.
Their idea is not new. In 1911, two American pathologists from the Pathological Laboratories, University of Pittsburgh, Pennsylvania, Oskar Klotz and M.F. Manning, published a summary of their studies of the human arteries and concluded that "there is every indication that the production of tissue in the intima is the result of a direct irritation of that tissue by the presence of infection or toxins or the stimulation by the products of a primary degeneration in that layer."25 Other researchers have presented similar theories.26
Researchers have proposed many potential causes of vascular injury, including mechanical stress, exposure to tobacco fumes, high LDL-cholesterol, oxidized cholesterol, homocysteine, the metabolic consequences of diabetes, iron overload, copper deficiency, deficiencies of vitamins A and D, consumption of trans fatty acids, microorganisms and many more. With one exception, there is evidence to support roles for all of these factors, but the degree to which each of them participates remains uncertain. The exception is of course LDL-cholesterol. Much research allows us to exclude high LDL-cholesterol from the list. Whether we look directly with the naked eye at the inside of the arteries at autopsy, or we do it indirectly in living people using x-rays, ultrasound or electron beams, no association worth mentioning has ever been found between the amount of lipid in the blood and the degree of atherosclerosis in the arteries. Also, whether cholesterol goes up or down, by itself or due to medical intervention, the changes of cholesterol have never been followed by parallel changes in the atherosclerotic plaques; there is no dose-response. Proponents of the cholesterol campaign often claim that the trials indeed have found dose-response, but here they refer to calculations between the mean changes of the different trials with the outcome of the whole treatment group. However, true dose-response demands that the individual changes of the putative causal factor are followed by parallel, individual changes of the disease outcome, and this has never occurred in the trials where researchers have calculated true dose-response.
A detailed discussion of the many factors accused of harming the arterial endothelium is beyond the scope of this article. However, the protective role of the blood lipids against infections obviously demands a closer look at the alleged role of one of the alleged causes, the microorganisms.
Is Atherosclerosis an Infectious Disease?

For many years scientists have suspected that viruses and bacteria, in particular cytomegalovirus and Chlamydia pneumonia (also named TWAR bacteria) participate in the development of atherosclerosis. Research within this area has exploded during the last decade and by January 2004, at least 200 reviews of the issue have been published in medical journals. Due to the widespread preoccupation with cholesterol and other lipids, there has been little general interest in the subject, however, and few doctors know much about it. Here I shall mention some of the most interesting findings.26
Electron microscopy, immunofluorescence microscopy and other advanced techniques have allowed us to detect microorganisms and their DNA in the atherosclerotic lesions in a large proportion of patients. Bacterial toxins and cytokines, hormones secreted by the white blood cells during infections, are seen more often in the blood from patients with recent heart disease and stroke, in particular during and after an acute cardiovascular event, and some of them are strong predictors of cardiovascular disease. The same is valid for bacterial and viral antibodies, and a protein secreted by the liver during infections, named C-reactive protein (CRP), is a much stronger risk factor for coronary heart disease than cholesterol.
Clinical evidence also supports this theory. During the weeks preceding an acute cardiovascular attack many patients have had a bacterial or viral infection. For instance, Dr. Armin J. Grau from the Department of Neurology at the University of Heidelberg and his team asked 166 patients with acute stroke, 166 patients hospitalized for other neurological diseases and 166 healthy individuals matched individually for age and sex about recent infectious disease. Within the first week before the stroke, 37 of the stroke patients, but only 14 of the control individuals had had an infectious disease. In half of the patients the infection was of bacterial origin, in the other half of viral origin.27
Similar observations have been made by many others, for patients with acute myocardial infarction (heart attack). For instance, Dr. Kimmo J. Mattila at the Department of Medicine, Helsinki University Hospital, Finland, found that 11 of 40 male patients with an acute heart attack before age 50 had an influenza-like infection with fever within 36 hours prior to admittance to hospital, but only 4 out of 41 patients with chronic coronary disease (such as recurrent angina or pervious myocardial infarction) and 4 out of 40 control individuals without chronic disease randomly selected from the general population.28
Attempts have been made to prevent cardiovascular disease by treatment with antibiotics. In five trials treatment of patients with coronary heart disease using azithromyzin or roxithromyzin, antibiotics that are effective against Chlamydia pneumonia,yielded successful results; a total of 104 cardiovascular events occurred among the 412 non-treated patients, but only 61 events among the 410 patients in the treatment groups.28a-e In one further trial a significant decreased progression of atherosclerosis in the carotid arteries occurred with antibiotic treatment.28f However, in four other trials,30a-d one of which included more than 7000 patients,28d antibiotic treatment had no significant effect.
The reason for these inconsistent results may be that the treatment was too short (in one of the trials treatment lasted only five days). Also, Chlamydia pneumonia, the TWAR bacteria, can only propagate inside human cells and when located in white blood cells they are resistant to antibiotics.31 Treatment may also have been ineffective because the antibiotics used have no effect on viruses. In this connection it is interesting to mention a controlled trial performed by Dr. Enrique Gurfinkel and his team from Fundación Favaloro in Buenos Aires, Argentina.32 They vaccinated half of 301 patients with coronary heart disease against influenza, a viral disease. After six months 8 percent of the control patients had died, but only 2 percent of the vaccinated patients. It is worth mentioning that this effect was much better than that achieved by any statin trial, and in a much shorter time.
Does High Cholesterol Protect Against Cardiovascular Disease?

Apparently, microorganisms play a role in cardiovascular disease. They may be one of the factors that start the process by injuring the arterial endothelium. A secondary role may be inferred from the association between acute cardiovascular disease and infection. The infectious agent may preferably become located in parts of the arterial walls that have been previously damaged by other agents, initiating local coagulation and the creation of a thrombus (clot) and in this way cause obstruction of the blood flow. But if so, high cholesterol may protect against cardiovascular disease instead of being the cause!
In any case, the diet-heart idea, with its demonizing of high cholesterol, is obviously in conflict with the idea that high cholesterol protects against infections. Both ideas cannot be true. Let me summarize the many facts that conflict with the idea that high cholesterol is bad.
If high cholesterol were the most important cause of atherosclerosis, people with high cholesterol should be more atherosclerotic than people with low cholesterol. But as you know by now this is very far from the truth.
If high cholesterol were the most important cause of atherosclerosis, lowering of cholesterol should influence the atherosclerotic process in proportion to the degree of its lowering.
But as you know by now, this does not happen.
If high cholesterol were the most important cause of cardiovascular disease, it should be a risk factor in all populations, in both sexes, at all ages, in all disease categories, and for both heart disease and stroke. But as you know by now, this is not the case
I have only two arguments for the idea that high cholesterol is good for the blood vessels, but in contrast to the arguments claiming the opposite they are very strong. The first one stems from the statin trials. If high cholesterol were the most important cause of cardiovascular disease, the greatest effect of statin treatment should have been seen in patients with the highest cholesterol, and in patients whose cholesterol was lowered the most. Lack of dose-response cannot be attributed to the knowledge that the statins have other effects on plaque stabilization, as this would not have masked the effect of cholesterol-lowering considering the pronounced lowering that was achieved. On the contrary, if a drug that effectively lowers the concentration of a molecule assumed to be harmful to the cardiovascular system and at the same time exerts several beneficial effects on the same system, a pronounced dose-response should be seen.
On the other hand, if high cholesterol has a protective function, as suggested, its lowering would counterbalance the beneficial effects of the statins and thus work against a dose-response, which would be more in accord with the results from the various trials.
I have already mentioned my second argument, but it can’t be said too often: High cholesterol is associated with longevity in old people. It is difficult to explain away the fact that during the period of life in which most cardiovascular disease occurs and from which most people die (and most of us die from cardiovascular disease), high cholesterol occurs most often in people with the lowest mortality. How is it possible that high cholesterol is harmful to the artery walls and causes fatal coronary heart disease, the commonest cause of death, if those whose cholesterol is the highest, live longer than those whose cholesterol is low?
To the public and the scientific community I say, "Wake up!"
Sidebars
Risk Factor

There is one risk factor that is known to be certain to cause death. It is such a strong risk factor that it has a 100 percent mortality rate. Thus I can guarantee that if we stop this risk factor, which would take no great research and cost nothing in monetary terms, within a century human deaths would be completely eliminated. This risk factor is called "Life."
Barry Groves, www.second-opinions.co.uk.
Familial Hypercholesterolemia - Not as Risky as You May Think

Many doctors believe that most patients with familial hypercholesterolemia (FH) die from CHD at a young age. Obviously, they do not know the surprising finding of the Scientific Steering Committee at the Department of Public Health and Primary Care at Radcliffe Infirmary in Oxford, England. For several years, these researchers followed more than 500 FH patients between the ages of 20 and 74 and compared patient mortality during this period with that of the general population.
During a three- to four-year period, six of 214 FH patients below age 40 died from CHD. This may not seem particularly frightening but as it is rare to die from CHD before the age of 40, the risk for these FH patients was almost 100 times that of the general population.
During a four- to five-year period, eight of 237 FH patients between ages 40 and 59 died, which was five times more than the general population. But during a similar period of time, only one of 75 FH patients between the ages of 60 and 74 died from CHD, when the expected number was two.
If these results are typical for FH, you could say that between ages 20 and 59, about 3 percent of the patients die from CHD, and between ages 60 and 74, less than 2 percent die, in both cases during a period of 3-4 years. The authors stressed that the patients had been referred because of a personal or family history of premature vascular disease and therefore were at a particularly high risk for CHD. Most patients with FH in the general population are unrecognized and untreated. Had the patients studied been representative for all FH patients, their prognosis would probably have been even better.
This view was recently confirmed by Dr. Eric Sijbrands and his coworkers from various medical departments in Amsterdam and Leiden, Netherlands. Out of a large group they found three individuals with very high cholesterol. A genetic analysis confirmed the diagnosis of FH and by tracing their family members backward in time, they came up with a total of 412 individuals. The coronary and total mortality of these members were compared with the mortality of the general Dutch population.
The striking finding was that those who lived during the 19th and early 20th century had normal mortality and lived a normal life span. In fact, those living in the 19th century had a lower mortality than the general population. After 1915 the mortality rose to a maximum between 1935 and 1964, but even at the peak, mortality was less than twice as high as in the general population.
Again, very high cholesterol levels alone do not lead to a heart attack. In fact, high cholesterol may even be protective against other diseases. This was the conclusion of Dr. Sijbrands and his colleagues. As support they cited the fact that genetically modified mice with high cholesterol are protected against severe bacterial infections.
"Doctor, don’t be afraid because of my high cholesterol." These were the words of a 36-year-old lawyer who visited me for the first time for a health examination. And indeed, his cholesterol was high, over 400 mg/dl.
"My father’s cholesterol was even higher," he added. "But he lived happily until he died at age 79 from cancer. And his brother, who also had FH, died at age 83. None of them ever complained of any heart problems." My "patient" is now 53, his brother is 56 and his cousin 61. All of them have extremely high cholesterol values, but none of them has any heart troubles, and none of them has ever taken cholesterol-lowering drugs.
So, if you happen to have FH, don’t be too anxious. Your chances of surviving are pretty good, even surviving to old age.
Scientific Steering Committee on behalf of the Simon Broome Register Group. Risk of fatal coronary heart disease in familial hypercholesterolaemia. British Medical Journal 303, 893-896, 1991; Sijbrands EJG and others. Mortality over two centuries in large pedigree with familial hypercholesterolaemia: family tree mortality study. British Medical Journal 322, 1019-1023, 2001.
From The Cholesterol Myths by Uffe Ravnvskov, MD, PhD, NewTrends Publishing, pp 64-65.
References
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Jacobs D and others. Report of the conference on low blood cholesterol: Mortality associations. Circulation 86, 1046–1060, 1992.
Iribarren C and others. Serum total cholesterol and risk of hospitalization, and death from respiratory disease. International Journal of Epidemiology 26, 1191–1202, 1997.
Iribarren C and others. Cohort study of serum total cholesterol and in-hospital incidence of infectious diseases. Epidemiology and Infection 121, 335–347, 1998.
Claxton AJ and others. Association between serum total cholesterol and HIV infection in a high-risk cohort of young men. Journal of acquired immune deficiency syndromes and human retrovirology 17, 51–57, 1998.
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Niebauer J and others. Endotoxin and immune activation in chronic heart failure. Lancet 353, 1838-1842, 1999.
Vredevoe DL and others. Skin test anergy in advanced heart failure secondary to either ischemic or idiopathic dilated cardiomyopathy. American Journal of Cardiology 82, 323-328, 1998.
Rauchhaus M, Coats AJ, Anker SD. The endotoxin-lipoprotein hypothesis. Lancet 356, 930–933, 2000.
Rauchhaus M and others. The relationship between cholesterol and survival in patients with chronic heart failure. Journal of the American College of Cardiology 42, 1933-1940, 2003.
Horwich TB and others. Low serum total cholesterol is associated with marked increase in mortality in advanced heart failure. Journal of Cardiac Failure 8, 216-224, 2002.
Elias ER and others. Clinical effects of cholesterol supplementation in six patients with the Smith-Lemli-Opitz syndrome (SLOS). American Journal of Medical Genetics 68, 305–310, 1997.
Bhakdi S and others. Binding and partial inactivation of Staphylococcus aureus a-toxin by human plasma low density lipoprotein. Journal of Biological Chemistry 258, 5899-5904, 1983.
Flegel WA and others. Inhibition of endotoxin-induced activation of human monocytes by human lipoproteins. Infection and Immunity 57, 2237-2245, 1989.
Weinstock CW and others. Low density lipoproteins inhibit endotoxin activation of monocytes. Arteriosclerosis and Thrombosis 12, 341-347, 1992.
Muldoon MF and others. Immune system differences in men with hypo- or hypercholesterolemia. Clinical Immunology and Immunopathology 84, 145-149, 1997.
Feingold KR and others. Role for circulating lipoproteins in protection from endotoxin toxicity. Infection and Immunity 63, 2041-2046, 1995.
Netea MG and others. Low-density lipoprotein receptor-deficient mice are protected against lethal endotoxemia and severe gram-negative infections. Journal of Clinical Investigation 97, 1366-1372, 1996.
Harris HW, Gosnell JE, Kumwenda ZL. The lipemia of sepsis: triglyceride-rich lipoproteins as agents of innate immunity. Journal of Endotoxin Research 6, 421-430, 2001.
Netea MG and others. Hyperlipoproteinemia enhances susceptibility to acute disseminated Candida albicans infection in low-density-lipoprotein-receptor-deficient mice. Infection and Immunity 65, 2663-2667, 1997.
Ross R, Glomset JA. The pathogenesis of atherosclerosis. New England Journal of Medicine 295, 369-377, 1976.
Ross R. The pathogenesis of atherosclerosis and update. New England Journal of Medicine 314, 488-500, 1986.
Klotz O, Manning MF. Fatty streaks in the intima of arteries. Journal of Pathology and Bacteriology. 16, 211-220, 1911.
At least 200 reviews about the role of infections in atherosclerosis and cardiovascular disease have been published; here are a few of them: a) Grayston JT, Kuo CC, Campbell LA, Benditt EP. Chlamydia pneumoniae strain TWAR and atherosclerosis. European Heart Journal Suppl K, 66-71, 1993. b) Melnick JL, Adam E, Debakey ME. Cytomegalovirus and atherosclerosis. European Heart Journal Suppl K, 30-38, 1993. c) Nicholson AC, Hajjar DP. Herpesviruses in atherosclerosis and thrombosis. Etiologic agents or ubiquitous bystanders? Arteriosclerosis Thrombosis and Vascular Biology 18, 339-348, 1998. d) Ismail A, Khosravi H, Olson H. The role of infection in atherosclerosis and coronary artery disease. A new therapeutic target. Heart Disease 1, 233-240, 1999. e) Kuvin JT, Kimmelstiel MD. Infectious causes of atherosclerosis. f.) Kalayoglu MV, Libby P, Byrne GI. Chlamydia pneumonia as an emerging risk factor in cardiovascular disease. Journal of the American Medical Association 288, 2724-2731, 2002.
Grau AJ and others. Recent bacterial and viral infection is a risk factor for cerebrovascular ischemia. Neurology 50, 196-203, 1998.
Mattila KJ. Viral and bacterial infections in patients with acute myocardial infarction. Journal of Internal Medicine 225, 293-296, 1989.
The successful trials: a) Gurfinkel E. Lancet 350, 404-407, 1997. b) Gupta S and others. Circulation 96, 404-407, 1997. c) Muhlestein JB and others. Circulation 102, 1755-1760, 2000. d) Stone AFM and others. Circulation 106, 1219-1223, 2002. e) Wiesli P and others. Circulation 105, 2646-2652, 2002. f) Sander D and others. Circulation 106, 2428-2433, 2002.
The unsuccessful trials: a) Anderson JL and others. Circulation 99, 1540-1547, 1999. b) Leowattana W and others. Journal of the Medical Association of Thailand 84 (Suppl 3), S669-S675, 2001. c) Cercek B and others. Lancet 361, 809-813, 2003. d) O’Connor CM and others. Journal of the American Medical Association. 290, 1459-1466, 2003.
Gieffers J and others. Chlamydia pneumoniae infection in circulating human monocytes is refractory to antibiotic treatment. Circulation 104, 351-356, 2001
Gurfinkel EP and others. Circulation 105, 2143-2147, 2002.

Kimberley
5th May 2012, 17:59
Thank you for posting Dr Uffe Ravnskov's brilliant article: "The Benefits of High Cholesterol"!!

James Yoseph is writing an essay each week to help promote the book and stir the pot.
Below is a copy for this weeks essay.



Doctors and Malpractice

Malpractice has become the rule of medicine.

How did this happen? Is it true? If it is, what can we do about it?

I have said “medicine has lost its way.” As I researched and listened to Hannah I began to be aware that if medicine ever had a way it was lost much earlier than I had thought.

Today there is no longer any value in seeking a second opinion. It will be the same opinion as the first. The protocols and treatment modalities are memorized and medicine is worse than cookie cutter it is production line and leaves no room for variation and true science.

Our doctors practice medicine by rote memorization of some one else’s idea of how medicine should be practiced. If it is wrong modality then every doctor practices the same wrong modality. Gradually the wrong modalities have become so numerous as to make malpractice the norm instead of the exception.

Doctors have become no more than trained parrots playing back the words they have been trained to say. Since no one wants to admit being wrong or be different than his or her peers, medicine either cannot change or if it does, change is very slow.

Medicine has deteriorated into arrogant ignorance.

The verities and excitement of science is lost to doctors and has been replaced with a 007 license to kill with impunity. Doctors are serial killers.

Do no harm has been replaced with do not be different than your peers. Do not question your drug rep, do not think for yourself, cover your ass and avoid being sued. And for God’s sake get all the paperwork in compliance with the insurance providers.

The open mindedness of true science has been replaced with the doctrine of double blind studies and insurance requirements, which are generated by people with a profit motive.

Doctors sit in their private dining room off the cafeteria and discuss their portfolios and how to get paid by the insurance carriers not whether what they are practicing might be wrong. No room for dissension in the private cafeteria unless it is about which stock is best for long term or short term investment.

It is a sickening and appalling truth.
Daniel Steinberg, in his own words, stamped out the last vestige of independent thinking being done by doctors.

He wrote that doctors must stop resisting his lipid theory and the new drugs (statins) he was promoting and he would see to it they did. And he did.

Whatever was left of thoughtful medical practice he (one man) eradicated in favor of power, prestige and money. No one knows how much he was paid by the drug companies but what is known is that over $300,000,000 tax payer dollars were used by him to accomplish the feat.

The residual is runaway malpractice. The ignorance of doctors is no excuse.

James B. Yoseph

Co-author “How Statin Drugs REALLY Lower Cholesterol” and Kill You One Cell at a Time

ThePythonicCow
5th May 2012, 19:24
25% of the population of 1st world countries take statin drugs!!
Kimberley also posted an earlier thread on this topic, at: IMPORTANT-How Statin Drugs REALLY Lower Cholesterol (And Kill You One Cell at a Time) (http://projectavalon.net/forum4/showthread.php?43560-IMPORTANT-How-Statin-Drugs-REALLY-Lower-Cholesterol--And-Kill-You-One-Cell-at-a-Time-).

Kimberley
7th May 2012, 13:08
I wonder if anyone has any questions or comments about this interview? I can easily get a reply from the Yoseph's.

nearing
7th May 2012, 17:42
I wonder if anyone has any questions or comments about this interview? I can easily get a reply from the Yoseph's.

Yes, thanks Kimberley. Can you ask them if a patient with familial hypercholesterolemia should be treated with anything?

pilotsimone
7th May 2012, 17:57
deleted post

Kimberley
8th May 2012, 01:11
Hey pilotsimone thank you!!!

Here is an e-mail with a listener on my terrestrial radio station in Boston... as the Statin interview aired there this Sunday...
*************

From: [email]thomas_dolan
Sent: Monday, May 07, 2012 9:40 AM
To: Jaeger, Kimberley
Subject: Sunday morning show on statins


Hi Kimberly -

I caught your show Sunday morning on statins - very alarming for me as I have been taking them for years and exhibit many of the symptoms of which they spoke. I was in the car at the time - would you please send me a transcript of the show and a link to where I can buy their book?

Thanks very much.

Regards,
Tom Dolan
*********


Hi Tom,

Sorry to hear about you having symptoms...however glad you heard the interview.

Here is a link for the interview on youtube...if you would prefer and mp3 version let me know.

http://www.youtube.com/watch?v=19uxXqWF8h4

And here are web site links for the information...

http://statininjuryvictims.blogspot.com/

http://statinsideeffects.webs.com/
"How Statin Drugs REALLY Lower Cholesterol (And Kill You One Cell at a Time)"


And here is a good article I recommend also:


Dr Uffe Ravnskov's brilliant article: "The Benefits of High Cholesterol" June 24 2004
http://www.westonaprice.org/cardiovascular-disease/benefits-of-high-cholesterol

All the best

Kimberley

Kimberley
9th May 2012, 12:18
**************************

Much love!!

cjbrooksjc
14th May 2012, 16:20
Science is learning that it is elevated LD(a) or small, sticky LDL cholesterol and inflammation that are the primary culprits behind CVIs ; not elevated cholesterol in general, and it is glucose-initiated inflammation first and foremost that precipitates the cascade. If you want to lower your potential for CVIs, lower your carb intake - particularly refined carbs and sugar. Statins not only reduce cholesterol levels dangerously, they damage the body's mitochondria and weaken muscle tissue (and your most important muscle is?...). Big Pharma wants to make us FEEL we are taking proper precautions; they do not want to make us well, and that is what is driving STATIN sales.

I have the subj. book and it is a winner. The Cholesterol Myths by Uffe Ravnvskov is great too but a bit pricey. There are two sites that have a wealth of info on the Statin dilemma: www.spacedoc.com/board/ (NASA flight surgeon) and http://stopped_our_statins.webs.com/ Both of these sites are filled with first-hand experiences.

Get smart - Get well

Brooks

nearing
14th May 2012, 17:48
Excellent post, Brooks. I couldn't agree more!

crested-duck
14th May 2012, 18:03
I've been off those killer pills for months now. Took them for little over a year after having a very expensive genetic blood test done for my cholesterol specifics. Started haveing frequent cramps in legs and occasional chest pains that made me question a possible stroke or heart attack happening, along with signs of frequent forgetfulness,and fatigue . I'm feeling better and better every day since stopped, sure am glad I figured it out sooner rather than later before too much damage was done.-Rob

Kimberley
16th May 2012, 19:09
:bump: :bump: :grouphug:

Kimberley
19th May 2012, 17:30
***************************************


May 18, 2012

Today, actually just minutes ago, new drug company generated studies were released that promote the good of statin drugs. Tell any lie loud enough and long enough and sooner or later nearly everyone will believe it.

STATINS ARE POISON. Absolutely, unequivocally, scientifically proven fact.

If you have taken statins drugs then you have been poisoned. That is what they do. Statin drugs are extracted from or synthesized to replicate fungal toxins. Toxins are called that for a reason. They are toxic.

Statins do not reduce or block cholesterol. They cause the transfer of cholesterol from the blood stream into cells. The blood cholesterol lowers. Cells die and are blocked from replicating. Cell replication is vital to life. Slow death begins immediately.

There is no such thing as a statin side effect. Statin poisoning is a direct and predictable effect of statin use.

Every statin user is a victim of this process. Some people have almost immediate symptoms.

The medical modality for statin-induced symptoms is to change statins rather than stop their use altogether.

Some people develop these effects slowly and are usually misdiagnosed as to the cause of their disease or discomfort.

The most common and immediate statin use effect is muscle soreness and pain.

The other effects as symptoms of statin use are to many to list but here are a few:


· Type 2 diabetes

· Arthritis

· Exacerbated CVD, heart attack and stroke

· High blood pressure caused by kidney damage

· Renal failure

· Cardio-myopathy resulting in low blood pressure and death

· Cancer especially breast and prostate cancer

· MS

· ALS Lou Gehrig’s disease

· GERD

· Diverticulosis

· Premature aging

· Mental disorders to include depression and rage.

· Loss of memory

· Cataracts

· Hearing loss

· Loss of balance

· Loss of libido in both men and women

· Erectile dysfunction

· Chronic fatigue

· ILD Interstitial lung disease

Statins are genotoxic and cause cell mutations.

Everyone taking statins long term will develop one or more of these direct effects of statin use.

There are two kinds of cell death. Necrosis is normal cell death. It is why cells replicate and replace themselves. Apoptosis is abnormal cell death caused by statins and other poisons.

Statins block cell replication and cause apoptosis.

Statins block the mevalonate pathway inside the cell. It is down that pathway that come all the ingredients (isoprenoids) essential to life, energy and health.

If you have permanent damage from statin use, buy “How Statin Drugs REALLY Lower Cholesterol” and take it to an attorney. It was written as a go by for civil litigation.

Some statin injuries are slow to repair (brain and muscle) and may never fully heal.

You have a right to remedy.

James B. Yoseph

foreverfan
20th May 2012, 04:51
I got off of them after 3 years about a month ago.
I was put on them after a heart attack and a 5 way bypass. I continued to feel like total crap. Finally my wife's chiropractor told us that statins blocked the lipid antioxidant CoQ10 and suggested I take 300mg if I was on the drug. After about a week I felt much better. The next time I went to my cardiologist I asked him why he hadn't told me this. He told me he didn't think this was important. I couldn't believe it. I wanted to punch the guy in the face. Needless to say, I fired him.

From what I've learned, heart disease is caused by inflammation.
The way to eliminate inflammation and to actually reverse heart disease is with antioxidants (vitamin C, Alpha Lopic-Acid & CoQ10), Revestarol, Magnesium, Vitamin D3 and 3 simple Amino Acids (L-Cartinine, L-Arginine & L-Citrulline).

Here is information on a doctor that actually cures people.
Dr. Joseph Prendergast “Father of the Year” for 2008 (http://www.rmsb.com/Main/Files/Dr_%20Joe.pdf)

http://www.healthywaysnow.com/images/wrs_prendergast.jpg

The ADA honors Dr. Joseph Prendergast for:

* Saving more lives than any other physician in the USA
* Clinically testing and documenting the results of more than 6,000 of his own patients
* Not admitting ONE patient to a hospital in 19 years
* Not losing ONE patient to Heart Attack or Stroke in 19 years


GREAT DOCS TO RESEARCH ONLINE
You can follow their stuff on Youtube and also check out Dr Steven Sinatra, Dr. Louis Ignarro & Dr. Robert Heaney. You won't be sorry you did.

DON'T MISS THIS ON FISH OIL & CANCER!!! Brian Peskin (http://www.youtube.com/watch?v=LxGj5Fd53n4&feature=relmfu)

After doing over 100 hours of research, I developed my own supplement plan.
I don't sell it. I have been on it for 2 years. It only costs about $25 per month. PM me if you are interested. I just tell you the formula, the links to the science behind it, and the best places to buy the supplements.

Kimberley
21st May 2012, 19:15
*************

Here is a great 9 minute history of today's pharmaceutical industry...

The Rockefellers, The FDA & The Cancer Industry

sDjcVrZnemM


This documentary explains the Rockefeller influence on the health care industry, and particularly how safe alternatives have been silenced in favor of chemotherapy, radiation and surgery. Going back to the early twentieth century, this movie explains how it all got started, and why we are in our current health care predicament. It then provides cures for those suffering with cancer.

Only when these "companys" stop getting funded, will we see an admission of ONE OF THE MANY CURES THAT ALREADY EXIST.

Fund them .... And you just fund somebodys job, while millions die.

Mike
21st May 2012, 20:27
*bump*

my Dad takes statins, and i've just recently convinced him to take coq10 along with them, thank God.

his Cardiologist mentioned to him that some cardiologists in Europe are obligated to make this suggestion to their patients. he told me this as if i hadn't already said it to him dozens of times;). with my Dad, if you're not wearing a white lab coat he will not take you seriously. but i notice he's giving my suggestions a little more gravity lately, so i'm pleased with that.

modwiz
21st May 2012, 20:41
At 59 I need no drugs, I take ibuprofen when massage/bodywork is not an option. I don't go to doctors for check-ups. I feel great and don't need a second opinion. I some point I will die at some point in the future and am ready for that. Enriching pharmaceutical companies on my way to that destination is not in my plans.

This works for me and it is as a contrast that it is offered. What we eat and think (mind being the most important, IMO) is the real secret to health. Your DNA is listening. What are you telling it?

Kimberley
25th May 2012, 03:15
The latest from James Yoseph... May 24, 2012


Doctors
Doctors simply do not know they have been duped.

The way they are taught in the world of competition and medicine is rigid unforgiving and tough. Their instructors teach them that there is only one way to practice. The amount of information they must absorb and memorize is astonishing.

They universally must go through a year of dissecting a cadaver one piece at a time and naming every nerve, vein and organ. It is not a place for dummies or the squeamish. Neither is med school a place for the slacker or lazy.

They memorize every symptom, protocol and modality of each and every ailment of a possible patient. They begin seeing patients in their 3rd year and ordering treatment and meds by the 4th. They move on to internship and work impossible long hours under incredible stress. At any given moment they can lose their chosen career for one mistake.

By the time they graduate they know everything they must know to practice medicine, as it is known today.

What is wrong with this picture?

The teaching of doctors is a closed system. It is the same everywhere in America.

It is antiquated, barbaric and wrong.

One or two wrong modalities and diagnosis become universally accepted and cannot be changed. Missed diagnosis and wrong information is perpetuated as if it were the law of the land.

If treatment becomes the “standard of care” instead of cure it is the same everywhere. If a bad drug makes it to market everyone prescribes it. Your doctor still lives in fear of any deviation in treatment. He could still lose his chosen career for refusing to ascribe to the rules. The result is an entire planet is poisoned with a pathogenic fungal toxin endorsed by physicians everywhere.

Is that the man or woman you want to see for a physical or minor ailment?

What is your alternative?

The tree is rotten at the root.

James B. Yoseph

foreverfan
25th May 2012, 03:27
Thanks to all who requested my protocal. Let me know how it goes for you. :thumb:

cjbrooksjc
27th May 2012, 18:54
This is a lengthy but informative supporting view from a practicing cardiologist:

**http://www.sott.net/articles/show/242516-Heart-Surgeon-Speaks-Out-On-What-Really-Causes-Heart-Disease

Remove the asterisks (**) before you paste the link; they discourage hitch-hiker spam.

Best,

Brooks

Kimberley
28th May 2012, 19:42
This is a lengthy but informative supporting view from a practicing cardiologist:

**http://www.sott.net/articles/show/242516-Heart-Surgeon-Speaks-Out-On-What-Really-Causes-Heart-Disease

Remove the asterisks (**) before you paste the link; they discourage hitch-hiker spam.

Best,

Brooks

Thank you Brooks, a great addition to this thread!

Also I got word today that the Yoseph's have a full web site now....

http://statinsideeffects.weebly.com/

Much love ! :grouphug:

Kimberley
29th May 2012, 02:03
Here is more.....

http://www.oneradionetwork.com/health/hannah-yoseph-md-and-james-b-yoseph-how-statin-drugs-really-lower-cholesterol-and-kill-you-one-cell-at-a-time-april-26-2012/

Kimberley
1st June 2012, 17:01
This belongs here...


ofZRm-kfggA

Kimberley
2nd June 2012, 18:04
I want to add this here also... less than 10 minutes...

The Rockefellers, The FDA & The Cancer Industry

ZbyY3jqvVBQ

Kimberley
10th June 2012, 15:15
This weeks essay. I wrote it in response to a continuing question I get from people who are frustrated with trying to help.

Love,

James

When God lay the foundation of the World, He Gave us two Laws that are perfect in their implementation and Justice.

Those are: For every action there is an equal and opposite reaction and an object set in motion stays in motion.

Those govern the physical universe, the mental and the spiritual. Those are immutable, unchangeable and permanent. As Sovereign of the universe He can change anything but He does not. Why would He change the perfect?

How difficult is it to change someone’s, anyone’s mind? Next to Impossible. Once a thought or belief is set in motion it becomes a moon encircling its planet, the larger the moon the greater its inertia.

Mentally and spiritually the only force great enough to change a mind or spirit is fear or pain with one exception.

The mind and spirit have their own inertia. A force great enough can change their direction but the law remains immutable. Only a force great enough can change the direction and spin of the moon. The same is true of our inner being.

That is what makes changing something as important as prescribing statins as medicine, so difficult. The wrong idea, the malevolent implementation not withstanding it is an object set in motion.

The Law is amoral. It simply is. Good or bad set in motion stay in motion until enough force comes along to change it.

The Truth is not enough of itself. In fact Truth without Love is not Truth at all. It becomes a bitter cruelty to be resisted and rightfully so.

The only force great enough to change anyone’s mind is the combination of Love and Truth. If we can love enough when we present the Truth it will move mountains. That is the only force greater than fear and pain.

There is not enough fear and pain to get the job done until it is too late. Intervention can be done early only in Love and Truth.

There are only three kinds of people who change their minds, those with open minds to begin with, those in enough pain and those who are loved enough.

James B. Yoseph

Kimberley
12th June 2012, 03:53
Yes I admit that I am bumping this again!!!!

:bump:

Kimberley
15th June 2012, 16:49
This weeks essay from James..

The Good of Statins

What we know about the direct effects of taking statin drugs is a potential boon to medicine. Statins are implicated in Cancer, ALS, MS, Chronic Fatigue, obesity, auto immune diseases, cardio myopathy, and a host of others to include exacerbated CVD (Cardiovascular disease) the disease they are supposed to prevent.

There is a huge body of irrefutable stats that have surfaced to prove just how deadly and harmful statins truly are.

If we can encourage medicine to pay attention we can learn a lot from statins as to the cause and effect relationship between mycotoxins and disease. Statins are mycotoxins.

This terrible man made plague has the potential to save lives.

I would like to sit before some senate oversight committee and ask three questions to be answered by a show of hands:

• How many of you are taking statins?
• How many of you are suffering what you consider to be some of the discouraging ravages of aging?
• How many of you trust your doctor implicitly?

If they told the truth, most of the hands would go up in answer to every question.

In the first human study done in the US a hand picked group of six people were selected to take statins. They were heterozygotes or people with the rare genetic proclivity to hypercholesterolemia. Four of the six had the expected precursors of CVD. Two did not. All were given a statin drug. As expected the cholesterol levels came down.

Doctors Brown, Goldstein, Bilheimer and Grundy headed up the program. They blithely walked past the most significant part of the study. Two, fully one third of the people selected, had no sign of CVD; that fact in spite of their rare genetic proclivity.

Men with an agenda have blinders on. They were there to study the effects of statins. It never occurred to them to study the remarkable health of the two. What was different about them? Instead they gave them a health-destroying drug.

If we do not begin studying health we will never be and become healthy. The study of medicine and drugs is upside down………………………………………….Study health.

James Yoseph June 15, 2012

eileenrose
16th June 2012, 03:39
My mom just came home from a 3 days hospital stay due to Statin drugs. Of course, the doctors there (she had five different ones...can you imagine that???!!!) didn't take her off of them. They just said there was nothing wrong that they could find. ie. don't bother us.

I assumed they would kick her out earlier, when they found nothing wrong, but they kept her an extra day (then I remembered she has complete medical coverage...she pays for that). so they kept her to make money off of her.

Losers!!!

Now that she is home, she is starting to question this particular drug and it's potential side effects.
...but I guess it isn't my worry (she just ignores me...I have to have some level of authority she trust...ie a m.d. degree, I suppose). What you print here, on PA, she thinks is all fantasy.

Life is short. love your parents while you still got them. That is my slogan. Because they do everything they are told to by the/their doctors. Even if they don't even know their names.
I should ask her, was it doctor number 1 or 2 or 3 or 4 or 5 that told you that it isn't your prescriptions that are killing you?

what an awful world we live in (right now). that is all I got to say on it.


...still stinging....from her refusal to see this (yet....but obviously, she will keep getting sick/hospilized...this isn't going away).

Kimberley
16th June 2012, 04:07
EileenRose... I hear and feel your pain... Buy the book and ask her to read it or read it to her and maybe she will see the light!! However what ever happens it is all in divine order!!!

Much love!

eileenrose
16th June 2012, 04:17
Kim- sent you a PM. I am not in a 'good' mood, on this topic (today). Still stings (that she ignored me and ended up there). My dad is starting to see that it might be them. Stay tuned. Let's see if we can steal back a life (from those bad bad boys...ie. contributing doctor's who perpuated the myth that statins are safe).

nearing
16th June 2012, 23:29
Another MD on the subject ---
(http://www.drbriffa.com/2012/06/15/statins-can-drain-the-life-out-of-us/)

Statin drugs reduce cholesterol by inhibiting the an enzyme in the liver (HMG-CoA reductase) which plays a role in the production of cholesterol in the liver. Unfortunately, this enzyme also plays a part in the production of a substance known as Coenzyme Q10, which itself is important for energy production within the body’s cells. Statins therefore have the ability to drain the life out of people. Any doctor who sees patients and actually listens to them will know this from experience, and now someone’s actually gone and shown it with a scientific study [1].

The study was published on-line in the Archives of Internal Medicine. A group of individuals were randomised to take one of two statins (simvastatin at 20 mg per day or pravastatin at 40 mg per day) or placebo for six months. Participants were rated at regular intervals through the study for their perceived fatigue on exertion, general fatigue and energy levels.

One thing worth highlighting here is that the study was only 6 months in duration. This is relevant because it’s not uncommon for the adverse side-effects of statins to come on many months or even years after the treatment is started.

Overall, statins did indeed appear to cause a significant change in energy and worsen fatigue on exertion. Women were more affected than men.

Four out of 10 women reported either reduction in energy or worsening of fatigue on exertion.

Two out of 10 women reported problems with both these things.

One out of 10 women reported that both of these things were ‘much worse’.



The authors remark:

Effects were seen in a generally healthy sample given modest statin doses, and both simvastatin and pravastatin contributed to the significant adverse effect of statins on energy and fatigue with exertion. Particularly for women, these unfavorable effects were not uncommon… These findings are important, given the central relevance of energy and functional status to well-being.

If you or someone you know appears to have statin-related fatigue or other symptoms (such as muscle pain), please see this blog post about how this might be reversed using supplements of Coenzyme Q10.

References:

1. Golomb BA, et al. Effects of Statins on Energy and Fatigue With Exertion: Results From a Randomized Controlled Trial. Arch Int Med epub 11 June 2012

eileenrose
17th June 2012, 02:35
Statin drugs reduce cholesterol by inhibiting the an enzyme in the liver (HMG-CoA reductase) which plays a role in the production of cholesterol in the liver. Unfortunately, this enzyme also plays a part in the production of a substance known as Coenzyme Q10, which itself is important for energy production within the body’s cells. Statins therefore have the ability to drain the life out of people.

yes, that is it. in a nutshell.

The Vampire Drug

---
Update:
6/17, So a couple more relatives have objected to Statin use (with my mother). Hopefully that does the trick. ...don't know if she has quit (yet).
6/18 she quit taking it. Hope that last (she wants the doctor to tell her what to do...so she's made an appointment...and when I say 'doctor'..I mean the random dude that she gets at the clinic she visits...they change almost yearly now...and each new one gives her more medications than the last.....

...ugg!.....people...will we ever ever wake up from this drug disaster???)

It is worse that just about any other. fyi.

Kimberley
20th June 2012, 17:48
This weeks essay from James...

I engaged in a brief Internet exchange with someone who identified himself as “foxholeathiest”. The language patterns and technique used to attempt to invalidate the findings in “How Statins Drugs REALLY Lower Cholesterol” reminded me of Jonathan Tobert. He as you know is one of the central villains in the conspiracy to bring statins to market.

It was typical of the kind of effort we expected to see from the drug cartels. It was the first all out frontal attack based not in fact but rather in side steps and obfuscation. He pretended to have not read the book and could not attack the content but rather attacked the motive for bringing it to market, accused us of fear tactics, money grubbing and attacked the manner of publishing. The book could not be valid and true because we self-published. He said people would die from not taking their statins.

Though we could have deleted all his comments and mine, I left them posted for public review.

I did not apologize or justify. For our readers, friends and supporters we need do neither.

What he wrote was indelicate subterfuge and drug company propaganda. That was what brought statins to market. Scare the hell out of everyone about his or her LDLs.
Then provide a poisonous toxin to assuage their fear. It has grown into a medical catastrophe of monumental proportion.

Not to worry. If we can engender more of this type of attack it will raise public awareness and we will have done our job. I had begun to be concerned that they might just ignore us.

The book continues to sell as we continue to write. “Poisoned” will soon be published. It started as a recovery and health tome for the injured and has grown into a sequel full of new facts and paradigm shifts.

“How Statin Drugs REALLY Lower Cholesterol” was not written for the layperson. It simply has too much difficult scientific fact that had to be included. It will not be a best seller nor will it make the authors rich and famous. It was written for the victims and their attorneys. It is a go by for civil litigation. In it is sufficient evidence to do battle in a court of law based on “failure to warn” which is what won the huge tobacco awards. The victims have a right to remedy.

The cure for statin injury is known. It is mevalonate. It will not come to market in our lifetimes. It would require and admission of guilt from the only people who could manufacture it, the drug companies. There is not enough morality in the entirety of them to ever make such an admission. It is sad but true.

If you are statin injured your only hope of remedy is good diet and lawsuit.

James Yoseph June 20, 2012

Kimberley
27th June 2012, 17:41
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This weeks essay from James...

We try to answer all emails. Two consistent themes come up that are tough to talk about. One is statin induced mental illness, depression, irritability and rage. The other is loss of libido, ED and impotence.

Beatrice Golomb documented the increase in both problems in statin users.

What complicates the issues even more is that doctors are not trained to recognize them as side effects and consequently treat them as aging. Anti-depressants and tranquilizers may help the first but often exacerbate the second. Testosterone levels are checked. Estrogen and Viagra are prescribed and the side effects get treated.

A common theme in our reader’s questions is “I have quit statins.” “What now”?

Stopping statins, good diet and moderate exercise will correct most of it. But what about the residual scars left on families and relationships by depression, irritability and rage. How do we deal with the insecurities left by the loss of femininity or manliness? Have our relationships survived? Did we lose a good job?

Can we afford to blame statins for some momentary lapse? What about personal responsibility?

The near impossible dilemmas these questions represent are perhaps the most difficult of statin side effects. We cannot afford to abrogate personal responsibility in favor of blame.

We can however accept that what has happened, has happened. None of us is defined as human by a temporary period of impotence or anger.

We can take responsibility for seeing this never happens again to our children, our grandchildren or us. We can get well informed and take charge of our own health and we must.

We can ask ourselves some very important questions. How did good diet and food supplements get relegated to the fringes of medicine? What is wrong in the system of things that allows for these historical catastrophes?

What changes will work? Can we throw out the bath water and not the baby?

If we do not take personal responsibility and act; then this will inevitably be a repeat occurrence.

What kind of world are we leaving to our children’s children?

James Yoseph June 27, 2012

nearing
30th June 2012, 16:42
I helped a pt of mine who had been on statins for 8 years (she is only 41) and who got diagnosed with 'fibromyalgia', lo and behold, 8 years ago! The poor thing has been miserable with pain all over and migraine headaches as well as the accompanying depression and anxiety for all of this time and not one of her doctors connected the dots!

Her marriage has been on the rocks for the past 2 years due to all of this. She was in tears with hope when I gave her this info. I really hope for her sake that the pain isn't permanent by this time.

Kimberley
1st July 2012, 20:26
*************

James Yoseph said he has recovered well...expecting the same for your friend!! :hug:




I helped a pt of mine who had been on statins for 8 years (she is only 41) and who got diagnosed with 'fibromyalgia', lo and behold, 8 years ago! The poor thing has been miserable with pain all over and migraine headaches as well as the accompanying depression and anxiety for all of this time and not one of her doctors connected the dots!

Her marriage has been on the rocks for the past 2 years due to all of this. She was in tears with hope when I gave her this info. I really hope for her sake that the pain isn't permanent by this time.

Kimberley
4th July 2012, 04:33
**************
This weeks essay from James...July 3, 2012

Mycoplasmas and their fungi like spores are found in all living things. They are the one life form that may be immortal. They have been found alive in 3,000-year-old mummies. Their spores make toxins. Some of those poisons are more virulent than others.

There is a vast body of knowledge about them that is just the tip of the iceberg. Much more remains to be discovered than is now known.

On our journey to discover the origins of statins we discovered a whole new world of information that is alien to medicine. In the medical field only veterinarians study and treat for micotoxicity in their patients. Yet these tiny creatures are implicated in a host of degenerative human diseases. There is much theory about them and their purpose. Some say they are like the rest of life in that they compete for various food sources and develop their toxins to kill off competing organisms.

Why would an immortal living creature feel the need to compete?

What is clear about them is, as they go through 16 known morphologies, they decay and degenerate their host organism. They are responsible for rot from the inside, the same way mold decays from the outside. In so doing they make all other life possible.

A world without mycoplasma and their spores would be devoid of life.

What becomes obvious is, their importance is irrefutable. Equally important is that all other living things naturally resist their disease causing decay. As the resistance wears down the decay begins and grows until the cycle is complete in death and degeneration.

Suddenly good health and nutrition is no longer important. It is paramount. Without maintaining and nurturing our body’s natural resistance, decay begins then accelerates.

Statins being the product of mycoplasmas, a mycotoxin, accelerate decay.

The study of disease is useless without the understanding of these organisms and sure and certain knowledge of what prevents them causing their genetic given to destroy.

Science must study health. What causes health? What preserves health? What restores health? Without the study of health, it will remain beyond our grasp.
All medicine should include the study of health and restoration.

Mycotoxins are not candidates for prevention; instead they are Pandora’s box.

James Yoseph Junly 3, 2012

Kimberley
4th July 2012, 04:42
******

Just found this also.... less than 8 minutes...

TZomZEPBdbM

Kimberley
16th July 2012, 15:11
******************

This weeks essay from James Yoseph...July 16, 2012

I received a question this morning from a severely injured statin victim.

His question was, “James, what are your thoughts on how to bring this evil to an end?”

My answer as follows:

I am not sure it can be. It is so entrenched that our whole culture is brainwashed into believing in it. I fear it will take a catastrophe of monumental proportions to get it done.

Nevertheless I hope to the contrary. If it is to stop then it requires keeping our credibility in place. With our available information we can document everything before going public. The critics can then only lie and obfuscate. Those tactics become transparent. The answer to them becomes ridicule.

I do not defend my position. If I am right, then no defense is required and being defensive undermines trust. I have nothing to defend. The facts speak for themselves.

I believe that if there is an answer it will be done with provable facts, going public then following through by continually stirring the pot. It will take bulldog determination and dedication. People, who try, typically are angry and frustrated. Those emotions burn out and they quit in discouragement. Satan wins.

It requires an inner work. We must be free of our own greed, lust for authority and anger. Those things are what got us in this mess in the first place. Revolutions begin in the hearts and minds of men. If they take place in the streets then we end up with a whole new set of bastards to dominate us.

If we can do that individually and collectively then Truth trumps lies and obfuscations every time. God wins.

Persistence, determination and living in the reality that it may not happen in our lifetime are what will make it happen. We must be in it for the long haul and keep a close rein on our impatience.

If our motive is to help our fellow man as opposed to bring down the powers that be, we can win this battle. The powers that be will fall under the weight of their own wrongs.

Buddha, The Christ, Gandhi, St Francis and Martin Luther King changed the world for the better. That is the only change I am interested in.

James Yoseph July 16, 2012

Kimberley
20th July 2012, 17:33
This weeks essay from James Yoseph...July 20, 2012

We are losing the best and brightest of our culture and society. Seventy five million Americans mostly middle aged and older are taking statin drugs. The vast majority of people in that group are being poisoned by a deadly mycotoxin.

We are corresponding with people all over the world who have been injured. The worst stories are those with the mentally debilitating effects of statin use. Depression, rage, loss of memory, global amnesia and delusional paranoia lead the list of these terrible consequences. The effects on family, jobs and relationships are worse than horrific.

That stains remain on the market is diabolical. It is not just greed that drives the people and companies behind them; it is indifference. The facts are in. The studies have been done. There is nothing but indifference that could keep this deadly plague in place.

The losses in productivity are astronomical. The medical cost of treating the diseases they cause or exacerbate is breaking the back of our nation and the world.

I fear we have let our reader down. We understated the case against this monstrous evil. It becomes very personal when we sit and weep for the hurting souls who have had this drug foisted off on them by their doctors, big pharma, the FDA , the NIH , the AMA and the rest of the cohorts in this debacle.

This insanity must stop.

We are starting a forum for statin injured victims similar in scope and purpose of askapatient.com There, will be posted all available info including books from any and all authors and a forum for all statin victims to share their experience and hope. All information will be reviewed by a mediator for its usefulness and appropriateness to the central topic of statin injuries. At this forum our books will be available for free to all who cannot afford them and for as long as we can afford to send them.

This forum will be the beginning of a central clearing house to gather information on the as yet unpublished true scope of statin injury. It will also serve as a place for people to seek legal counsel and be linked to other helpful sites as people may wish.

Today, we received another arrogant letter from a Harvard MD who had not read the book but knew it was bunk anyway. He said we obviously did not know what we were doing and about all the new studies that were coming out in favor of statins. That is the problem exactly. We do know about all the new bogus studies that continue to come out in contradiction of the facts.

Again, this madness must stop

James Yoseph July 20, 2012

ljwheat
20th July 2012, 17:59
******

Just found this also.... less than 8 minutes...

TZomZEPBdbM

Great find Kimberly, what about using this greed you were talking about in a different why or direction.

A lawyer or law firm hungry for money would gain in a global or local, class action law suite, just like they do with asbestos and tobacco adds on TV. Billions of dollars to be paid out, hit the drug companies below the belt. John xoxo

nearing
26th July 2012, 02:21
"Your Homocysteine level is a more accurate predictor than cholesterol of our risk of heart attack or stroke." (http://members.upnaway.com/~poliowa/homocysteine%20unveiled.html)

"Homocysteine reflects the health of your genes." say Patrick Holford and Dr James Braly MD in their 2003 publication "The H Factor". "Your H score is more important than your weight, your blood pressure or your cholesterol level. It is your most vital, preventable and reversible health statistic." and "Your Homocysteine level is a more accurate predictor than cholesterol of our risk of heart attack or stroke."

So why aren't doctors looking at it? They can. And YOU can ask your GP to do the blood test - a fasting plasma homocysteine. The Labs will give "normal levels" as 6.5 - 11.9mmol/L or even higher but Halford recommends a level below 6.

Clinipath results here in WA come back with a notation for the doctor that says - "Elevated Homocysteine levels are associated with increased independent risk of cardiovascular disease. A 5 mmol/L rise in HoCy correlates to a 0.5mmol/L increase in Cholesterol. Elevations are also found in anaemia, genetic homocystinuria, renal disease, deficiencies of B group vitamins B12, Folate, Pyridoxine (ie B6) and some drug therapies."

Royal Perth Hospital began a study about 4-5 years ago on the effectiveness of using B12, B6 and folic acid to lower homocysteine levels. This was replicating previous similar studies already undertaken in other parts of the world. I'm not sure what stage this present study at Royal Perth Hospital has reached but we don't need to wait.

The new book by Holford, brings in some more essential nutrients that are needed for these same chemical pathways to occur. We, the public, have not been told of these additional essential co-factors - namely B2, zinc, magnesium and choline (or TMG).
HOMOCYSTEINE is a naturally occurring by-product of methionine metabolism in the body. Methionine is an amino acid, ie part of food proteins and is found primarily in meats, eggs, dairy products, fish, chicken, seeds, nuts and some vegetables. Homocysteine pathways normally pave the way to body production of other essentials including glutathione a powerful detoxifier in the body on the one hand and brain chemicals serotonin (the happy hormone), melatonin (sleep and mood improving hormone), dopamine (euphoria hormone) and adrenaline (the fight and flight hormone).

According to Holford, the reason Homocysteine accumulates in the body causing cell damage and the onset of major disease, is because the biochemical transformation process is not working properly, usually due to lack of these needed vitamins and minerals for the given Homocysteine pathways.

In this diagram, protein foods like meats in the diet breakdown to methionine then to homocysteine. Normally it then goes on to fulfil various important pathways to provide essential hormones etc. (SAMe is a metabolic precursor to the essential brain hormones.) If we are short on the needed vitamins and minerals for these pathsways we end up with dangerous excess homocysteine levels and ill health.

So as you can see we do need homocysteine to get the end-result good things like serotonin, adrenaline, melatonin and dopamine and to detoxify in the liver but unless the other vitamin and mineral co-factors are present we end up building dangerous and damaging levels of homocysteine in the body. But we can fix this - take the appropriate supplements, have your GP test your H levels and get them down to 6.

Why You Need to Have Your Homocysteine Levels Checked (http://www.liveinthenow.com/article/why-you-need-to-have-your-homocysteine-levels-checked)

Many doctors limit their cardio screening process to a cholesterol and triglyceride test, despite the fact that other factors have been shown to contribute significantly to the development of heart disease. More Americans die each year from heart disease than from any other cause. This fact alone should make it standard practice to use every screening tool available to detect possible risk factors associated with heart disease. Cardiac specific c-reactive protein is a marker of inflammation in the blood vessels, now thought to be a serious risk factor in heart disease. In addition to c-reactive protein, there is a blood test that can reveal important information about a person’s risk level. This test measures levels of a blood marker called homocysteine.

What Is Homocysteine?

Homocysteine is a naturally occurring amino acid produced in our body by a process of conversions carried out during various metabolic cycles. In most people it does not stick around for long. Shortly after being synthesized, the body quickly metabolizes it back into a non-harmful amino acid. However, for various reasons some individuals do not efficiently recycle homocysteine, resulting in elevated blood levels of this potentially harmful amino acid.

In 1968, a Harvard researcher observed that children with a specific genetic disorder that predisposed them to elevated homocysteine levels had a 50% increased risk of death due to vascular disease. He also observed that using interventions to lower homocysteine levels decreased this risk. He summarized that elevated homocysteine levels could serve as an independent risk factor for a person’s likelihood of developing vascular disease.

Further research has confirmed his theory many times over. The most recent evidence of this was published in the Mayo Clinic Proceedings Journal in November of 2008. It evaluated the outcome of 26 other studies related to how homocysteine levels affect cardiovascular risk. The study was unique in that it was the first one to only use data from individuals who had never had coronary disease before. This is in response to two recent studies showing no benefit for B vitamin therapy among patients with established coronary heart disease. Having pre-existing coronary disease might mask potential benefits of lowering homocysteine using folic acid and other B vitamins.

The results of the meta-analysis were impressive. It showed that every 5-point elevation in blood homocysteine levels resulted in a 20% increase risk for the development of coronary heart disease. This increase in risk is totally independent of any other factors that influence coronary heart disease development, which makes it a very significant risk factor in and of itself.

What Is Normal?

Normal – 5 to 15 µmol/L
Moderate – 16 to 30 µmol/L
Intermediate – 31 to 100 µmol/L
Severe – Above 100 µmol/L
There are many reasons why a person may have elevated homocysteine levels. To understand some of these reasons it is important to explain the process our body uses to naturally keep homocysteine levels low. Homocysteine is recycled into two other amino acids called methionine and cysteine. For this process to occur, some very key nutrients are needed. Folic acid, vitamin B6 and vitamin B12 are the primary nutrients needed to recycle homocysteine into non-harmful amino acids.

A large percentage of the American population has elevated homocysteine levels. This is likely due to a poor diet devoid of the critical nutrients necessary for the conversion of homocysteine into its inert amino acid counterparts. Fast food and heavily processed foods can rob the body of important nutrients. Research has shown that switching to a diet that contains foods rich in folic acid and other B vitamins can reduce homocysteine levels. Dark leafy greens like spinach, kale and collard greens typically have the highest levels of folic acid. Whole grains like brown rice and other fruits and vegetables typically have high levels of vitamin B6. B12 is most commonly found in animal products. Some great sources include wild salmon, dairy products like yogurt and cheese, and eggs.

The process of aging also creates an increased risk of elevated homocysteine levels due to decreased intestinal absorption of folic acid and other B vitamins. As we age, our digestive secretions such as stomach acid, bile acids and pancreatic enzymes decrease. This reduces the ability of our body to break down and extract nutrients from our foods.

A small percentage of the American population has elevated homocysteine levels due to a genetic abnormality. The methylenetetrahydrofolate reductase enzyme (MTHFR) is the enzyme responsible for converting homocysteine into methinonine. The genetic abnormality affects the activity of this enzyme. Up to 10% of the population may have some form of this genetic irregularly and never know it unless tested properly. People with elevated homocysteine levels have a much higher risk of forming blood clots. This is of particular concern for people taking certain medications. Women who form blood clots while taking oral birth control pills or hormone replacement often have this genetic abnormality. Because the formation of blood clots can lead to life threatening conditions such as a pulmonary embolism, heart attack or stroke it should be standard practice to test for these genetic abnormalities before giving medications that might increase the risk of a potential problem. Patients receiving potent drugs for chemotherapy are also at a greater risk of forming clots if they have the MTHFR deficiency. Ten percent is a very high number when looking at the relative rate of a genetic abnormality in the general public.

Why Is Homocysteine Bad for Me?

Although it is not fully understood, there are several possible mechanisms that are responsible for homocysteine’s negative effect on the cardiovascular system.

The initial trigger responsible for the initiation of plaque formation in an artery is injury. The delicate layer of endothethial cells that line our arteries are subject to damage from high blood pressure, toxins from alcohol, pollution and cigarette smoke and free radical damage. When that endothethial lining becomes damaged, the body is prompted to fix it by coating the damaged area with cholesterol. If other factors in the body are just right, that deposition of cholesterol can cascade into the formation of an artery blocking plaque. Homocysteine is thought to be one of the unique compounds capable of causing the initial damage to the delicate lining of arteries and thus triggering the cascade of plaque formation.

Another mechanism by which homocysteine can facilitate cardiovascular disease is related to its ability to make your platelets more likely to stick together. This increases the risk of clot formation and perpetuates the formation of arterial plaques.

So How Can I Fix It?

The first step is to identify if this is an issue for you. Asking your primary care doctor for a simple blood test to measure homocysteine is the first step. If elevated, there are several natural vitamins that can lower homocysteine levels.

What Nutrients Should I Consider?

The most important are going to be folic acid, B12 and B6. All three play a crucial role in the enzymatic steps necessary to convert homocysteine into its benign amino acid counterparts.

Folic acid is one of the most important nutrients for lowering homocysteine levels. It is the primary nutrient used by the body for converting homocysteine into methionine. Doses anywhere from 1 mg to 10 mg are commonly used and seen to create significant drops in homocysteine levels in most individuals.

B12 also plays an important role in the recycling process. It is most effective when given in its activated form often referred to as methylcobalamin. The more common form called cyanocobalamin has to be activated by the body before it can help with homocysteine metabolism making it less effective. B12 is best given as an injection or in a sublingual (dissolves under the tongue) lozenge.

B6 plays an important role in the metabolism of homocysteine into something called cysteine. This is another recycling pathway the body has for homocysteine and works greatly in our favor because cysteine is a direct precursor to the potent antioxidant called glutathione. Glutathione plays a critical role in helping our body eliminate toxins via the liver and serves as the most potent circulating antioxidant. Again, it is recommended to use the activated form of B6 (pyridoxine-5 phosphate) to maximize effectiveness.

Make sure to have your doctor check your blood homocysteine levels at your next visit. If your levels are high, adding folic acid, B12, and B6 to your supplement regimen is almost guaranteed to lower elevated homocysteine levels. And due to the absence of virtually any side effects and little known toxicity issues, this is a low risk way to greatly reduce a major risk factor for heart disease.

Warlock
30th July 2012, 04:11
My wife's doctor tried to put her on statins and when I objected, I became "Public Enemy #1".

She gave my wife some samples of that junk and while I told my wife not to take them, she tried them for two days and got an awful headache, so she stopped.

That was about two years ago.

I told my wife to eat more oatmeal in the morning. Lo' and behold, her last test showed her cholesterol levels down. I don't know if that was the cause, but as long as she doesn't take that crap, she'll be fine.

Warlock

boja
29th August 2012, 18:56
Headlines in today's UK "Daily Mail" (One of the UK's biggest) :-

GIVE STATINS TO ALL OVER-50s

"Statins should be prescribed to all over-50s regardless of their health history, a leading British expert said yesterday"

so says Sir Rory Collins (an Oxford University Professor), followed by a whole page article.

OK, so who's right, and who's wrong ???

JAMES YOSEPH versus SIR RORY COLLINS

(Please don't shoot me, I'm just the messenger).

nearing
29th August 2012, 20:48
Headlines in today's UK "Daily Mail" (One of the UK's biggest) :-

GIVE STATINS TO ALL OVER-50s

"Statins should be prescribed to all over-50s regardless of their health history, a leading British expert said yesterday"

so says Sir Rory Collins (an Oxford University Professor), followed by a whole page article.

OK, so who's right, and who's wrong ???

JAMES YOSEPH versus SIR RORY COLLINS

(Please don't shoot me, I'm just the messenger).

Does this Sir Rory back up his proposal with physiological science? Does he have ties to Big Pharma? Does he take them himself?

ThePythonicCow
29th August 2012, 22:16
Does this Sir Rory back up his proposal with physiological science? Does he have ties to Big Pharma? Does he take them himself?
He just might take statins himself. That might account for his limited mental acuities.

I see similar affects in dentists who recommend mercury fillings :).

nearing
30th August 2012, 02:17
Does this Sir Rory back up his proposal with physiological science? Does he have ties to Big Pharma? Does he take them himself?
He just might take statins himself. That might account for his limited mental acuities.

I see similar affects in dentists who recommend mercury fillings :).

Excellent point! Lol

sheddie
30th August 2012, 04:53
A close Friend has just stopped taking statins because he was in so much pain that he could hardly walk, the pains started to disappear as soon as he stopped taking them. He was borderline diabetic and borderline cholesterol levels ......so the Dr put him on statins and diabetic drugs, just in case!

He is on Metaformin for his diabetes and has had diarrhea for the last 3 weeks, he is weak and has lost a lot of weight and is still too scared to come off the diabetics drugs because he thinks something serious might happen to him!!!! He is not overweight and was fit and well before the medical profession got a hold of him.

What will it take for some people to wake up. It's very sad to see someone who was fit and well reduced to this state ......just in case he might get what???? A few simple diet changes would have done the trick but oh no they want you to take their drugs.

I dispair, my brother-in-law has come off statins after seeing the video as he was having such bad side effects and a another close friend is still on statins and complaining of leg pain amongst other things .... What will it take for people to realise just how bad these things are.

All of them have pretty bad diets and from speaking to them would rather fix their problems by taking tablets rather that make the effort to change their lifestyle......cos why should they?

Their attitude is very much like a another friend who has bowl cancer and said that there was no way he was changing his diet as in his words "I have eaten these foods for all of my life I am not going to change now" ..... hello, how did he think bowel cancer showed up, the clues are there......... he has been through so much pain, so many operations and yet absolutely refuses to even consider changing his diet in any way ....his diet is appalling ...... sorry rant over :o

Oh and for anyone not in the UK, the Daily Mail is not the most reliable paper around .....they will sensationalise to get their point across - just my opinion obviously but just thought I would put that out there :eyebrows:

fifi
6th October 2012, 00:33
My friend stopped taking Lipitor (a kind of statin drug) after I told him about this and showed him the video. But after 3 months stopping taking the drug, he did a test of his cholesterol level, and was alarmed that his Triglicerides number has trippled. He is considering taking Lipitor again. Can anyone give my friend an advice of how to lower his cholesterol level without taking statin drugs? He also takes Coq10, wonders if that helps.

Thanks a lot,
Fifi

nearing
6th October 2012, 19:13
My friend stopped taking Lipitor (a kind of statin drug) after I told him about this and showed him the video. But after 3 months stopping taking the drug, he did a test of his cholesterol level, and was alarmed that his Triglicerides number has trippled. He is considering taking Lipitor again. Can anyone give my friend an advice of how to lower his cholesterol level without taking statin drugs? He also takes Coq10, wonders if that helps.

Thanks a lot,
Fifi

Google Chris Kresser for his blog then search there for Cholesterol.

In the meantime, your friend should cutting out ALL carbs (including sugar) and polyunsaturated fats.

Rex
18th February 2013, 15:19
"Your Homocysteine level is a more accurate predictor than cholesterol of our risk of heart attack or stroke." (http://members.upnaway.com/~poliowa/homocysteine%20unveiled.html)

I recently read the book THE HOMOCYSTEINE SOLUTION by Patrick Holford and Dr. James Braly (as mentioned in your post) because I had read a mention of it in one of David Icke's books. After reading the incredible number of things that appear to be cured by keeping a super-low homocysteine level (a LONG list), a couple of them really struck me: 1) it basically allows DNA to repair itself, and 2) it allows proper melatonin production from the infamous pineal gland!

About 2 weeks ago I started a homocysteine vitamin regimen, and have some promising results so far:

1-Energy and drive is up
2-Healing is faster
3-Sleep is sounder

That said, I have to temper that by saying it's not like I suddenly feel 20 years younger. But there is definitely an effect, and you can take that from a guy who's tried many different vitamins and supplements over the years. Will keep you posted.