COVID19 – the spike protein and blood clotting

[Tintin]

Apologies in advance for yet another Covid thread but I feel that what Dr. Malcolm Kendrick is sharing here needs to be up-front and not buried in any extant threads.

This may just be about one of the very best summaries of what Covid-19 actually is and it's in my view pretty irrefutable.

It combines medical terminology (of course, no brainer) but very helpful plain English 'translations' that should make this really well understood to a wider non-medical audience.
.
It's here:
https://drmalcolmkendrick.org/2021/0...mpression=true and presented in full, below.

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COVID19 – the spike protein and blood clotting

Dr. Malcolm Kendrick
June 3rd, 2021

When COVID19 came along I was in the midst of writing my latest book on heart disease. What causes it – and what does not.

One section I was working on covers the wide range of conditions known as the vasculitis(es). I could immediately see a whole series of connections between COVID19, spike proteins, the immune system and blood clots. Some of which are deeply concerning, for reasons that should become apparent.

Before getting started, you can see an immediate problem here is there does not seem to be a plural form of vasculitis. A bit like octopus. You can have one octopus, but what happens then… two octupuses… or is it two octopi? Wars have been fought over less.

Anyway, a vasculitis is a condition whereby a factor, of some sort, causes damage to the vascular system. The vascular system being, essentially, the blood vessels and the heart. The suffix itis simplymeans inflammation. As in appendicitis, or tonsillitis. Or, in this case vasculitis.

There are many different vasculitis(es) or vasculiti? They range from Kawasaki’s disease to antiphospholipid syndrome, rheumatoid arthritis, scleroderma, Sjogren’s disease and suchlike. They are many, and varied, and quite fascinating. At least they are, to me.

In all of them you have two things in common… that are most relevant to this discussion. First, with any form of vasculitis, the body decides to attack the lining of the blood vessels – causing inflammation and damage. Second, the rate of death from cardiovascular disease goes up dramatically. In some cases, a fifty-fold increase. This was seen in young women with Systemic Lupus Erythematosus (SLE) with additional antiphospholipid syndrome1.

Why does the body decide to attack itself? This is a good question that I cannot really answer. If I could, I would be claiming my Nobel prize, right now. However, I can say that, for various reasons, the immune system makes the decision that it doesn’t like something about the lining of the blood vessels and believes it to have become ‘alien’ in some way. It then proceeds to attack. Which does not answer the question as to exactly why the attack happens? But it does tell you a bit about what happens.

Another major problem with vasculitis is that blood clots spring to life throughout the vascular system. This is because the blood is always ready to clot, at any time, and if you take away some of vital the anti-clotting mechanisms, the balance will be tilted firmly towards coagulation.

One of the most powerful anti-clotting mechanisms/systems is the protective layer that lines your entire vascular system, known as the glycocalyx. This is made up of glycoproteins (glucose and proteins stuck together). Under an electron microscope the glycocalyx looks like a tiny forest, or a badly mown lawn.

Many fish are covered with glycocalyx, which makes them very slippery, and difficult to get hold of. The glycocalyx also stops bacteria and viruses from gaining entry, in both fish and humans.

In your blood vessels, the glycocalyx protrudes out from endothelial cells, the cells that line all your blood vessels, and into the bloodstream. The layer of glycocalyx contains many, many, anticoagulant factors. Below is a short list of all the things the glycocalyx does:

The glycocalyx:

- Forms the interface between the vessel wall and moving blood.
- Acts as the exclusion zone between blood cells and the endothelium.
- Acts as a barrier against leakage of fluid, proteins and lipids across the vascular wall.
- Interacts dynamically with blood constituents.
- Acts as the “molecular sieve” for plasma proteins.
- Modulates adhesion of inflammatory cells and platelets to the endothelial surface.
- Functions as a sensor and mechano-transducer of the fluid shear forces to which the
endothelium is exposed; thus, the glycocalyx mediates shear-stress-dependent nitric
oxide production.
- Retains protective enzymes (e.g., superoxide dismutase).
- Retains anticoagulation factors, e.g.: Tissue factor inhibitor, Protein C, Nitric Oxide
(NO), Antithrombin.

Complicated stuff – that hardly anyone has ever heard of.

Anyway, if you damage the glycocalyx, or damage the underlying endothelial cells that synthesizes the glycocalyx layer, you will tip the balance very strongly towards the creation of blood clots. These can then then stick to the artery, or vein, wall. Sometimes they will fully block a blood vessel, leading to such things as a stroke or heart attack.

The interaction between vasculitis and thrombosis has been a relatively unexplored area of medicine. But it remains critically important in many diseases:

‘The relationship between inflammation and thrombosis is not a recent concept, but it has been largely investigated only in recent years. Nowadays inflammation-induced thrombosis is considered to be a feature of systemic autoimmune diseases such as Systemic Lupus Erythematosus (SLE), Rheumatoid Arthritis (RA), or Sjogren’s Syndrome (SS)2.

In super-short version. If you damage the lining of blood vessel walls, blood clots are far more likely to form. Very often, the damage is caused by the immune system going on the attack, damaging blood vessel walls, and removing several of the anti-clotting mechanisms.

Sepsis

Moving sideways for a moment. There are other things that can damage the blood vessel wall, leading to widespread blood clot formation. One of them is the condition known as sepsis. Which used to be called blood poisoning.

In sepsis, bacteria gain entry to the bloodstream through such things as a cut, an insect bite, a severe urine infection, and suchlike. When bacteria get into the blood, and start multiplying, they release exotoxins. Which are, effectively, the waste products of the bacteria.

These exotoxins then attack blood vessel walls, damaging the glycocalyx and endothelial cells. This drives the formation of blood clots throughout the body. The medical term for this is disseminated intravascular coagulation (DIC) = widespread blood clots in the vascular system.

The attacks not only cause clots, they can also cause the smaller blood vessels to weaken and burst. Which is why one sign of an infection with the meningococcal bacteria (the one that causes meningitis), is a rash. The rash is made up of dark, almost black, bruises. Once these start to appear, things are very bad. Potentially fatal, it means blood vessels are under severe attack and are breaking apart. Creating both bleeding and clots.

In truth, the ‘rash’ in meningitis is not really a rash at all. It is a sign of underlying, severe, vasculitis. The individual small bruises can also be called petechiae. Just to be scientific.

Another sign of widespread blood vessel damage, with the formation of multiple blood clots, is that the level of platelets in the bloodstream falls dramatically. For those who have never heard of such things, platelets are small cells that float about in the bloodstream. Their primary role is to co-ordinate the blood clotting system. If a red blood cell was the size of the Earth, a platelet would be about this size of the Moon.

If there is damage to blood vessels, platelets fling themselves at the area, and stick together to form a solid plug. They also release chemicals and enzymes that cause fibrin to be formed. Fibrin is the long sticky strand of protein that binds clots tightly together. Platelets also drag in red blood cells, and suchlike to make bigger and tougher clots. They have been called the conductors of the clotting orchestra.

In the process of doing all of these things, the number of platelets starts to fall. This is not surprising, as they are being used up to make blood clots/thrombi. Which means that one sign of widespread clot formation is a fall in the level of platelets (thrombocytopenia). This reliable sign of widespread coagulation, or disseminated intravascular coagulation (DIC).

Time for a quick re-cap.

What do we know?

What we now know, on the journey towards COVID19, are three important things.

- If you damage the endothelial cells/glycocalyx, blood clots will form and stick to the
side of blood vessels.
- Damage is often caused by immune system attack.
- Falling platelet levels are a sign of widespread blood clotting.

COVID19

What do we know about COVID19? First, it can only enter cells that have a receptor known as the angiotensin II receptor (ACE2 receptor). Cells with these receptors are mainly found in the lining of the lungs, and endothelial cells that line all blood vessels. Also, the epithelial /endothelial cells than line the intestines. If a cell does not have an ACE2 receptor, COVID19 simply cannot gain entry.

This was known years ago, when SARS-CoV was identified, the precursor of SARS-Cov2. Here from a paper in 2004:

‘The most remarkable finding was the surface expression of ACE2 protein on lung alveolar epithelial cells and enterocytes of the small intestine. Furthermore, ACE2 was present in arterial and venous endothelial cells and arterial smooth muscle cells in all organs studied. In conclusion, ACE2 is abundantly present in humans in the epithelia of the lung and small intestine, which might provide possible routes of entry for the SARS-CoV. This epithelial expression, together with the presence of ACE2 in vascular endothelium, also provides a first step in understanding the pathogenesis of the main SARS disease manifestations3.’

So, SARS-CoV gets into the body through the lungs and bowels. These are the places where the virus can gain access because it is where ACE2 receptors can mainly be found. Of course, SARS-Cov2 gets into the body in exactly the same way.

What happens once SARS-Cov2 gets into cells? Well, it does what all viruses do. It takes over various cellular mechanisms and forces the cell to produce more SARS-CoV2 viruses. This then kills, or severely damages those cells. This mainly occurs when ‘virions’ start to escape from within the cell. This damages the cell membrane, and in some cases can cause the cell to burst apart.

Essentially, SARS-Cov2 starts by damaging endothelial cells in the lungs, because it usually arrives here first. Fluid is released, and there is the breakdown of small blood vessels in the lungs, and the small airways. In this situation, the lungs begin to fail, and oxygen levels in the blood can fall dramatically.

Infection can also cause diarrhoea, as the epithelial cells in the intestines are damaged. To quote from ‘the COVID19 symptoms’ study:

‘We think COVID-19 causes diarrhoea because the virus can invade cells in the gut and disrupt its normal function 4.’

As far as I know, no-one has died of COVID19 diarrhoea. However, COVID19 can create such severe lung damage that people have died from respiratory failure or lung damage… call this form of disruption what you will. However, many/most people survive this phase.

It is what happens next that that kills the majority of people who become severely infected.

What happens next is that SARS-Cov2 gets into the bloodstream. It then invades endothelial cells, also pericytes and myocytes in the heart. Both of which have a high level of ACE2 receptors. Both of which are kind of vital for heart function 5,6.

Then…

What we now have is a major widespread vasculitis on our hands, with severe endothelial cell damage and disruption and damage to the glycocalyx. Blood clots, blood clots, blood clots, everywhere.

‘Coronavirus disease 2019 (COVID-19) causes a spectrum of disease; some patients develop a severe proinflammatory state which can be associated with a unique coagulopathy and procoagulant endothelial phenotype. Initially, COVID-19 infection produces a prominent elevation of fibrinogen and D-dimer/fibrin(ogen) degradation products. This is associated with systemic hypercoagulability and frequent venous thromboembolic events. The degree of D-dimer elevation positively correlates with mortality in COVID-19 patients. COVID-19 also leads to arterial thrombotic events (including strokes and ischemic limbs) as well as microvascular thrombotic disorders (as frequently documented at autopsy in the pulmonary vascular beds). COVID-19 patients often have mild thrombocytopenia* and appear to have increased platelet consumption, together with a corresponding increase in platelet production.7’

*a low level of platelets

The spike protein

Then, of course, we have the spike protein to consider. If this is the thing that the immune system recognises and attacks – which it almost certainly is – then cells which are growing SARS-Cov2 inside them, which then express the spike protein on their surface as the virions escape, will be identified as ‘the enemy’.

At which point, the immune system will start to attack the endothelium (and glycocalyx) in an attempt to wipe out the virus. This will tend to happen two or three weeks after the initial infection (sometimes sooner). This is after the immune system has had a real chance to identify the spike protein, then properly wind itself up to produce antibodies against it. This is the time of maximum attack on the endothelium.

This moment is often referred to as a cytokine storm. A point where every system in the immune system gets revved up and charges into action. At one point I wasn’t sure if I really believed in the cytokine storm. But I do now think it is a real thing. It is almost certainly why steroids (which very powerfully reduce the immune response) have been found to reduce mortality in severely ill patients.

All of which means it may well be the body’s own infectious disease defence system that creates much of the damage to the cardiovascular system. Not necessarily the virus itself.

Alternatively, it may be that the spike protein itself creates most of the blood clots. Here from the paper ‘SARS-CoV-2 spike S1 subunit induces hypercoagulability.’

‘When whole blood was exposed to spike protein even at low concentrations, the erythrocytes (red blood cells) showed agglutination, hyperactivated platelets were seen, with membrane spreading and the formation of platelet-derived microparticles8.’

Translation. Introduce SARS-CoV2 spike proteins into bloodstream, and it makes it clot – fast. Which is a worry.

Vaccines

It is a worry because the entire purpose of vaccination against SARS-Cov2 is to force cells to manufacture the spike protein(s) and then send them out into the bloodstream.

So, quick recap again, what do we know?

We know that a very high percentage of the people who die following a COVID19 infection, die as result of blood clots. We also know that they can also suffer severe myocarditis (inflammation of the heart muscle), and suchlike.

We know that the spike protein can stimulate blood clots all by itself.

We know that the immune system attack on ‘alien’ proteins, such as the spike protein, can cause vasculitis.

We know that vaccines are designed to drive the rapid production of spike proteins that will enter the blood stream specifically to encounter immune cells, in order to create a powerful response that will lead to ‘immunity’ against future SARS-CoV2 infection.

We know that a number of people have died from blood clots following vaccination. To quote from the European Medicines Agency website report on the AZ COVID19 vaccine:

The PRAC (pharmacovigilance risk assessment committee) noted that the blood clots occurred in veins in the brain (cerebral venous sinus thrombosis, CVST) and the abdomen (splanchnic vein thrombosis) and in arteries, together with low levels of blood platelets and sometimes bleeding 9.’

This was all pretty much predictable, if you understood what was going with SARS-CoV – nearly seventeen years ago.

My concern at this point is that, yes, we have identified very rare manifestations of blood clotting: cerebral venous sinus thrombosis (CVST) and splanchnic (relating to the internal organs or viscera) vein thrombosis (SVT). These are so rare that it is unlikely that anything else – other than a novel vaccine – could have caused them. I have never seen a case and I had never even heard of them before COVID19 came along. And I have spent years studying the blood coagulation system, and vasculitis, and suchlike.

So, if someone is vaccinated, then has a cerebral venous sinus thrombosis, or a splanchnic vein thrombosis, this is almost certainly going to be noted and recorded – and associated with the vaccination. Fine.

However, if there is an increase in vanishingly rare blood clots, could there also be an increase in other, far more common blood clots at the same time. If this was the case, then it would be far more difficult to spot this happening.

Millions and millions of people suffer strokes and heart attacks every year. Millions more suffer deep vein thrombosis and pulmonary emboli. In fact, around the world, tens of millions die each and every year as a result of a blood clots forming somewhere in the body.

That is a hell of a lot of background blood clotting noise. Which means that it could be extremely difficult to disentangle cause and effect, especially if you are not looking. If an elderly person is vaccinated, then dies of a stroke a couple of weeks later. What caused the blood clot that led to the stroke? It is unlikely that any doctor would record this as a post-vaccine adverse event.

To give you one example of the difficulty of disentangling cause and effect, when you are looking at very common events, a few years ago Merck launched a drug called Vioxx (an anti-inflammatory like ibuprofen, or naproxen but not exactly the same class of drug). It didn’t go well. Here from the article ‘Merck Manipulated the Science about the Drug Vioxx.’

‘To increase the likelihood of FDA (Food and Drug Administration) approval for its anti-inflammatory and arthritis drug Vioxx, the pharmaceutical giant Merck used flawed methodologies biased toward predetermined results to exaggerate the drug’s positive effects. Internal documents made public in litigation revealed that a Merck marketing team had developed a strategy called ADVANTAGE (Assessment of Differences between Vioxx And Naproxen To Ascertain Gastrointestinal tolerability and Effectiveness) to skew the results of clinical trials in the drug’s favor.

As part of the strategy, scientists manipulated the trial design by comparing the drug to naproxen, a pain reliever sold under brand names such as Aleve, rather than to a placebo.’

The scientists highlighted the results that naproxen decreased the risk of heart attack by 80 percent, and downplayed results showing that Vioxx increased the risk of heart attack by 400 percent. This misleading presentation of the evidence made it look like naproxen was protecting patients from heart attacks, and that Vioxx only looked risky by comparison. In fact, Vioxx has since been found to significantly increase cardiovascular risk, leading Merck to withdraw the product from the market in 2004.

Tragically, Merck’s manipulation of its data—and the FDA’s resulting approval of Vioxx in 1999—led to thousands of avoidable premature deaths and 100,000 heart attacks.’

https://www.ucsusa.org/resources/merck-manipulated-science-about-drug-vioxx

Yes, not exactly their finest hour. However, the point that I want to highlight from this sorry tale is that it is estimated that Vioxx caused 100,000 additional heart attacks, in the US alone, and nobody noticed. This figure was only worked out when researchers analysed the figures on increased risk, that had been seen in the clinical trials – at least the figures that were finally seen when Merck were forced to release the data.

You may think. How could one hundred thousand heart attacks simply be missed? Well, there are very nearly one million physicians in the US. If the heart attacks caused by Vioxx were evenly distributed, only one in five physicians would have seen anyone suffer because of taking Vioxx. In those physicians that did see one, or two, would they have made the connection? No, they would not. Not in a million years. There would not even be a record of any possible connection made.

Elderly person has a stroke, or heart attack. Elderly person took Vioxx. And…?

All of which means I am not gigantically concerned about CVST and SVT. Blood clots in these veins are rare, and remain rare, even after vaccination – and will never be missed, particularly when they happen in younger people. Because when younger people die, great efforts are made to establish the cause of death.

However, I can see no reason why these specific blood vessels would be targeted by blood clots. Perhaps there is some reason why clots only occur in the central venous sinus vein, or splanchnic vein following vaccination. If so, I have been unable to find out. I am more than willing to be educated on this.

Time to move on to the other worrying observation, that can be found within the report by the pharmacovigilance risk assessment committee (PRAC) – as mentioned above:

The PRAC noted that the blood clots occurred in veins in the brain (cerebral venous sinus thrombosis, CVST) and the abdomen (splanchnic vein thrombosis) and in arteries, together with low levels of blood platelets and sometimes bleeding.’

I shall finish here. You can join the dots yourself. Or not.

1: https://www.intechopen.com/books/pre...ltant-clinical

2: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4399148/

3: https://pubmed.ncbi.nlm.nih.gov/15141377/

4: https://covid.joinzoe.com/post/covid-symptoms-diarrhoea

5: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2614534/

6: https://academic.oup.com/cardiovascr...6/1097/5813131

7: https://www.karger.com/Article/FullText/512007

8: https://www.news-medical.net/news/20...ulability.aspx

9: https://www.ema.europa.eu/en/news/as...lots-low-blood

[onawah]

VAXXED Patients' Blood Examined, Horrific Findings Revealed by German Physicians
12690 views
8/20/21

https://www.brighteon.com/c4993b07-7...a-528709561a49

Source: https://www.brighteon.com/embed/c4993b07-754b-4a37-849a-528709561a49

"BREAKING! The 'Stew Peters Show' has obtained footage and slides of multiple patients' blood that was examined after being inoculated with the shots being called 'vaccines' for COVID-19.

Dr. Jane Ruby joins Stew Peters to discuss what the slides show, and how the blood is being changed, which can only be described as unexplainable and alarming."

https://rumble.com/vldaex-vaxxed-pat...ters+Show&ep=2


Source: https://www.rumble.com/video/vir49n

[meat suit]

copy and paste from Mercola, sorry video didnt paste,
https://articles.mercola.com/sites/a...rid=1241841014


Source: https://www.bitchute.com/video/xblF5p5Cul7H


Story at-a-glance

The FDA can only grant emergency use authorization for a pandemic drug or vaccine if there’s no safe and effective preexisting treatment or alternative. Since there are several such alternatives, the FDA is legally required to revoke the emergency authorization for these shots
While the COVID injections have been characterized as being somewhere around 95% effective against SARS-CoV-2 infection, this is the relative risk reduction, which tells you very little about its usefulness. The absolute risk reduction is only around 1% for all currently available COVID shots
Antibody-dependent enhancement (ADE) refers to a condition where the vaccination augments your risk of serious infection. We are now starting to see evidence that ADE is occurring in the vaccinated population
One of the most common side effects of the COVID shots is abnormal blood clotting, which can result in strokes and heart attacks
Even microclots that don’t completely block the blood vessel can have serious ramifications. You can check for presence of microclots by performing a D-dimer blood test. If your D-dimer is elevated, you have clotting somewhere in your body

In this interview, German microbiologist Dr. Sucharit Bhakdi sifts through the facts and fictions of the coronavirus pandemic. Together with Karina Reiss, Ph.D., he’s written two books on this subject, starting with “Corona False Alarm? Facts and Figures,” published in October 2020, followed by “Corona Unmasked: New Facts and Figures.”

The second book is currently only available in German, but you can download a free chapter of “Corona Unmasked” in English on FiveDoves.com.
Bhakdi’s Medical Credentials

Bhakdi graduated from medical school in Germany in 1970. After a year of clinical work, he joined the Max Planck Institute of Immunobiology, where he remained for four years as a post-doc.

There, he also began researching immunology. Eventually, he ended up chairing the department of medical, microbiology and hygiene at the University of Mainz, where he worked for 22 years until his retirement nine years ago. During that time, Bhakdi also worked on vaccine development, and says he’s “certainly pro-vax with regards to the vaccinations that work and that are meaningful.”

Much of his research focused on what’s called the complement system. When activated, the complement system ends up working in such a way that it destroys rather than aids your cells. Interestingly enough, SARS-CoV-2 uses this very system to its advantage, turning your immune system toward a path of self-destruction.

The same self-destructive path also appears to be activated by the COVID shots, which is part of why Bhakdi believes they are the greatest threat humanity has ever faced. “It is our duty to aggressively inform people about the dangers that they are subjecting themselves and their loved ones to by this ‘vaccination,’” he says.
How Effective Are the COVID Shots?

While the COVID injections have been characterized as being somewhere around 95% effective against SARS-CoV-2 infection, this claim is the product of statistical obfuscation. In short, they’ve conflated relative risk reduction and absolute risk reduction. The absolute risk reduction is actually right around 1% for all currently available COVID shots.1

In "Outcome Reporting Bias in COVID-19 mRNA Vaccine Clinical Trials"2 Ron Brown, Ph.D. calculates the absolute risk reduction for Pfizer’s and Moderna’s injections, based on their own clinical trial data, so that they can be compared to the relative risk reduction reported by these companies. Here’s a summary of his findings:

Pfizer/BioNTech vaccine BNT162b2 — Relative risk reduction: 95.1%. Absolute risk reduction: 0.7%
Moderna vaccine mRNA-1273 — Relative risk reduction: 94.1%. Absolute risk reduction 1.1%

In a July 1, 2021, commentary in The Lancet Microbe,3 Piero Olliaro, Els Torreele and Michel Vaillant also argue for the use of absolute risk reduction when discussing vaccine efficacy with the public. They too went through the calculations, coming up with the following:

Pfizer/BioNTech — Relative risk reduction: 95%. Absolute risk reduction: 0.84%
Moderna — Relative risk reduction: 94%. Absolute risk reduction: 1.2%
Gamaleya (Sputnic V) — Relative risk reduction: 91%. Absolute risk reduction: 0.93%
Johnson & Johnson — Relative risk reduction: 67%. Absolute risk reduction: 1.2%
AstraZeneca/Oxford — Relative risk reduction: 67%. Absolute risk reduction: 1.3%

What Kind of Protection Do the COVID Shots Provide?

Aside from providing insignificant protection in terms of your absolute risk reduction, it’s important to realize that they do not provide immunity. All they can do is reduce the severity of the symptoms of infection. According to Bhakdi, they fail even at this.

“They showed absolutely zero [benefit in the clinical trials],” he says. “This is the ridiculousness. People don't understand that they're being fooled and have been fooled all along. Let's take the one of these Pfizer trials: 20,000 healthy people were vaccinated and another 20,000 people were not vaccinated.

And then they observed, over a period of 12 weeks or so, how many cases they found in the vaccinated group and how many cases they found the non-vaccinated. What they found was that less than 1% of the vaccinated group got COVID-19 and less than 1% in the non-vaccinated group also got COVID-19.

The difference was 0.8 to 0.1%, which is nothing, considering the fact that they were not even looking at severe cases. They were looking at people with a positive PCR test — which as we all now know is worthless — plus one symptom, which could be cough or fever.

That is not a severe case of COVID-19. Any vaccination that is going to get authorized must be shown to protect against severe illness and death, and this has definitely not been shown. So, forget authorization. It can't be authorized, not by any normal means.

Now [the COVID injections do not have] full authorization, it's an emergency authorization, which again is absolute bull****, since we know the infection fatality rate of this disease or virus is not greater than that of seasonal flu. John Ioannidis has published these numbers, which have never been contested by anyone in the world and cannot be contested.

If you are under 70 years of age and have no severe preexisting illness, you can hardly die [from SARS-CoV-2 infection]. So, there is no fatality rate that can be reduced.

And for people who are elderly and have preexisting illness, as we know from Dr. Peter McCullough and his colleagues' work, there are very good means and medicines to treat this virus so that the fatality rates go down another 70 to 80%, which means there is no ground for emergency use whatsoever.

This means the FDA should be able to be forced to retract this emergency use authorization — unless they are in league with whoever wants to do this.”

I neglected to follow-up on his comment about 40,000 people being equally divided between the injection and no injection groups in the COVID injection trials. A few months ago, they actually abandoned the non-injection arm of the trial, so no there is no control group anymore.

The justification was that the injection was too important to deny it to the control group. It’s just another sneaky way to skirt around reporting all the adverse effects occurring in the injection group.

That said, it’s worth repeating that the FDA can only grant emergency use authorization for a pandemic drug or vaccine if there’s no safe and effective preexisting treatment or alternative. Since there are several such alternatives, the FDA is legally required to revoke the emergency authorization for these shots.
Evidence of Increased Infection Risk After Injection

Presently, the Centers for Disease Control and Prevention claims some 95% of SARS-CoV-2 infections resulting in hospitalization are occurring among the unvaccinated. This too is a statistical fiction, as they’re using data from January through June 2021, when most of the American public were unvaccinated.

Looking at more recent data, we’re finding that the majority of severe cases and hospitalizations are actually occurring among those that received the COVID jab. Unfortunately, as noted by Bhakdi:

“It's all manipulated. And, if someone wants to manipulate something and are in a position to then propagate it, you have no chance of analyzing it and telling people because we have no voice in this affair. When we stand up and tell people this, they just turn around and say that's not the truth.”

Disturbingly, we’re now starting to see the first indications of antibody-dependent enhancement (ADE), which many scientists were concerned about from the very beginning. India, for example, where 10% of the population has been “vaccinated,” is now seeing very severe cases of COVID-19. Bhakdi says:

“What we're witnessing in India and probably also in Israel is the immune dependent enhancement of disease … It's bound to happen. So, the people who are getting vaccinated now have to be fearful of the next wave of genuine infections, whether it's [SARS-CoV-2 variants] or any other coronaviruses, because they're all related and they will all be subject to immune dependent enhancement, obviously.”

Antibody-dependent enhancement (ADE), or paradoxical immune enhancement (PIE) refers to a condition where the vaccination results in the complete opposite of what you’re looking for. Rather than protect against the infection, the vaccine augments and worsens the infection.

ADE can occur through more than one mechanism, and Bhakdi is of the opinion that the enhancement is primarily due to over-reactive killer lymphocytes and secondary complement activation, both of which cause severe damage.
Antibodies Versus Lymphocytes

Bhakdi explains:

“There are two major arms of defense against viral infection. One is the antibodies that, if they are present, may prevent the virus from entering your cells. These are so-called neutralizing antibodies, which the vaccination is supposed to [produce].

But the antibodies are not at the place that they are needed, which is on the surface of the airway epithelium. They are in the blood, but not at the surface of the epithelium where the virus arrives. The second arm of immune defense then comes into play, and these are the lymphocytes.

There are different types of lymphocytes and I will simplify matters by saying the important lymphocytes are the so-called killer lymphocytes that sense whenever a virus product is being produced in the cell. They will then destroy the cells that harbor the virus and thus the factory is closed and you get well again.

That is the mechanism for how we can survive viral infections of the lung, and this happens all the time. So, the lymphocytes, in contrast to the antibodies, recognize many, many, many parts of the proteins. So, if a virus changes a little bit, it doesn't matter, because the waste products that are recognized by the killer lymphocytes remain very similar.

That is why all of us, and this is now known, all of us have memory lymphocytes in our lymph nodes and lymphoid organs that are trained to recognize these coronaviruses. And whether or not a mutant is there, it doesn't really matter, because they will recognize a mutant or variant.”

According to Bhakdi, coronaviruses can only undergo point mutations, meaning only one nucleotide at a time can be changed. The influenza virus, meanwhile, can undergo more radical mutations. For example, a flu virus can completely change its spike protein by swapping spike proteins with another virus that is simultaneously present.

This sort of shift is not possible with coronaviruses. Therefore, you will never have leaps in antigenic changes either for antibodies or for T-cell killer lymphocytes. That’s why the background immunity that evolves during the lifetime of a human being is very broad and solid.
Natural Immunity Is Far Superior to Vaccine-Induced Immunity

One of the most egregious nullifications of medical scientific truth is the claim that COVID “vaccination” confers superior protection compared than the natural immunity you get after you’ve been exposed to the virus and recover. The reality is that natural immunity is infinitely more superior to the vaccine-induced protection you get from these shots, which is both narrow and temporary.

The COVID shot produces antibodies against just one of the viral proteins, the spike protein, whereas natural immunity produces antibodies against all parts of the virus, plus memory T cells. As noted by Bhakdi:

“The very fact that the World Health Organization has changed the definition of herd immunity … is such a scandal. I'm at a loss of words to describe how ridiculous I find this all, that this is being accepted by our colleagues. How can the physicians and scientists of the world bear to listen to all this nonsense?”

How the COVID Shot Causes Damage

As explained by Bhakdi, when you get a COVID shot, genetic instructions are being injected into your deltoid muscle. Muscle drains into your lymph nodes, which in turn can enter your bloodstream. There may also be direct translocation from the muscle into smaller blood vessels.

Animal data submitted by Pfizer to Japanese authorities show the mRNA appeared within the blood within one or two hours of injection. The rapidity of it suggests the nano particles are translocated from the muscle directly into the blood, bypassing the lymph nodes.

Even microclots that don’t completely block the blood vessel can have serious ramifications. You can check for presence of microclots by performing a D-dimer blood test. If your D-dimer is elevated, you have clotting somewhere in your body.

Once inside your bloodstream, the genetic instructions are delivered to the cells available, namely your endothelial cells. These are the cells that line your blood vessels. These cells then start producing spike protein, as per the mRNA instructions. As the name implies, the spike protein looks like a sharp spike protruding from the cell wall, into the bloodstream.

Since they are not supposed to be there, your killer lymphocytes rush to the area, thinking the cells are infected. The killer lymphocytes attack the cells, which causes damage to the cell wall. This damage, in turn, provokes clot formation. We’re now seeing evidence that COVID shots are causing all manner of clotting issues, from microsized clots to massive clots stretching a foot or more in length.

Of course, when a large enough clot occurs in the heart, you end up with a heart attack. In the brain, you end up with stroke. But even microclots that don’t completely block the blood vessel can have serious ramifications. You can check for presence of microclots by performing a D-dimer blood test. If your D-dimer is elevated, you have clotting somewhere in your body.
How Vaccine-Induced Antibodies Can Cause Harm

But that’s not all. The anti-spike protein antibodies can also be harmful. Bhakdi explains:

“The other thing that has now emerged is just as frightening [as the clotting problem]. One to two weeks after the first jab, you start making antibodies in large amounts.

Now, when the second jab is done, and the spike proteins starts to project from the walls of your vessels into your bloodstream, it is not only met by the killer lymphocytes, but now the antibodies are also there and the antibodies activate [the] complement [system].

That was my first field of research. The first cascade system is the clotting system. Turn it on and the blood will clot. If you turn on the complement system with the antibodies that bind to your vessel wall, then this complement system will start creating holes in the vessel wall.

And you see these patients who have bleeding in the skin. Ask, where does that come from? Well, if you go around riddling your vessels with holes, you [get bleeding]. If the holes riddle vessels of the liver, or the pancreas or the brain, then the blood will seep through the vessels into the tissues …

[The COVID injections] are in your bloodstream for at least a week, and they will seep into any organ. And when those [organ] cells then start to make the spike protein themselves, then the killer lymphocytes will also seek and destroy them [in that organ, creating more damage and subsequent clotting].

What we are witnessing is one of the most fascinating experiments that could lead to massive autoimmune disease. When this will happen, God knows. And what this will lead to, God knows.”

COVID Jab May Trigger Latent Viruses and Cancer

The COVID jabs can also decimate your lymph nodes, as your lymph nodes are full of lymphocytes and other immune cells. Some of the lymphocytes will die immediately upon contact, causing inflammation.

Cells that don’t die and take up the mRNA and start producing spike protein will be recognized as virus producers and get attacked by the complement system. It essentially creates a war between some immune cells against other immune cells. As a result of this attack, your lymph nodes swell and become painful.

This is a serious problem, as the lymphocytes in your lymph nodes are lifelong sentinels that keep latent infection such as shingles under control. When they malfunction or are destroyed, these latent viruses can activate. This is why we’re seeing reports of shingles, lupus, herpes, Epstein-Barr, tuberculosis and other infections emerge as a side effect of the shots. Of course, certain cancers can also be affected.

“As we all know, tumors are forming every day in our bodies, but those tumor cells are recognized by our lymphocytes and then they're snuffed out,” Bhakdi says. “So, I am worried sick that the world is being goaded into taking something into the body that is going to change the whole face of medicine.”

Informed Consent Is Virtually Impossible

After giving this issue a great deal of thought, Bhakdi is convinced that the COVID injection campaign must be stopped.

“Gene-based vaccines are an absolute danger to mankind and their use at present violates the Nuremberg codex, such that everyone who is propagating their use should be put before tribunal,” Bhakdi says.

“Especially the vaccination of children is something that is so criminal that I have no words to express my horror … We are horribly worried that there's going to be an impact on fertility. And this will be seen in years or decades from now. And this is potentially one of the greatest crimes, simply one of the greatest crimes imaginable …

As we all know, it is laid down by the Nuremberg codex that in case experiments are to be conducted in humans, this can only be performed with informed consent.

Informed consent means that the person to be vaccinated has to be informed about all the risks, the risk benefit ratios, the potential dangers and what is known about side effects. This cannot be done with children, because children are not in the position to understand it.

Therefore, they cannot give informed consent. Therefore, they cannot be vaccinated. If anyone does that, he should be set before a tribunal. If grownups have been informed and want to get the shot, that's all right. But don't force anyone to get the shot. It has to be by informed consent only.”

Of course, informed consent is also virtually impossible even for adults, as they’re only given one side of the story. All side effects and risks are censored virtually everywhere and discussions about them are banned. The U.S. government is even pushing to criminalize discussion about COVID injection risks.
Where Do We Go From Here?

If you’ve already gotten one or two shots, there’s nothing you can do about that. Certainly, do not get a booster, as each booster is undoubtedly going to magnify the damage.

“In the end, I predict that we're going to see mass illnesses and deaths among people who normally would have wonderful lives ahead of them,” Bhakdi says. The question on people’s minds is, can anything be done to reverse the damage from these shots? As yet, we do not know.

However, if you have received one or more shots and develop symptoms of an infection, Bhakdi recommends treatment with hydroxychloroquine and/or ivermectin, such as the Zelenko protocol,4 and the MATH+ protocols,5 which have proven their effectiveness. It’s important to realize you may actually be more prone to serious infection, not less.

Nebulized hydrogen peroxide can also be used for prevention and treatment of COVID-19, as detailed in Dr. David Brownstein’s case paper6 and Dr. Thomas Levy’s free e-book, “Rapid Virus Recovery.” Whichever treatment protocol you use, make sure you begin treatment as soon as possible, ideally at first onset of symptoms.

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[Apulu]

Thanks for posting Tintin that was a really worthwhile read I think and it probably does deserve it's own thread. This could be the heart of the problem, in terms of affects on health, so to speak. Apologies for the pun.

I wonder could system-wide vascular problems be the reason for l"ong covid"? I haven't seen any research directly addressing that. Anyone?

It seems many people are being convinced to take the shot because they reason that long covid is a terrible, terrible thing affecting young and healthy people. And, low and behold... It doesn't appear the shot is going to help with that does it? Oh dear.

Goodness me people. My heart is really feeling all of this right now. It's getting hard to stay balanced, but... I will I hope. In the past few weeks it's becoming far more apparent where I am how many people have just gone along with this and I'm.... Just sad. And occasionally pretty angry. Deep breaths... And yoga.... What a ride. To say the least.

[jaybee]

Thank you for the opening post info, Tintin..... very educational and interesting (this is the only post I've read in this thread so far although I've already seen the Stew Peters / Jane Ruby video...)

While I was going through it a thought came to me...... ASPIRIN.....

How come we never hear anything/much about aspirin now...? Well I haven't noticed anything...

Could aspirin be something important that is being quietly ignored because it is even more accessible and cheaper than hyroxychloroquine and ivermectin.........?


And even if 'they' got it took off the shelves the bark of the willow tree could be used and was where aspirin came from in the first place....

just a couple of random links bringing this subject together ie aspirin with the OP...

https://www.mayoclinic.org/diseases-...y/art-20046797

Aspirin interferes with your blood's clotting action. When you bleed, your blood's clotting cells, called platelets, build up at the site of your wound. The platelets help form a plug that seals the opening in your blood vessel to stop bleeding.

But this clotting can also happen within the vessels that supply your heart with blood. If your blood vessels are already narrowed from atherosclerosis — the buildup of fatty deposits in your arteries — a fatty deposit in your vessel lining can burst.

Then, a blood clot can quickly form and block the artery. This prevents blood flow to the heart and causes a heart attack. Aspirin therapy reduces the clumping action of platelets — possibly preventing a heart attack.

http://edition.cnn.com/2010/HEALTH/1...ory/index.html

The word "aspirin" wasn't a coincidence. It comes from Spiraea, a biological genus of shrubs that includes natural sources of the drug's key ingredient: salicylic acid. This acid, resembling what's in modern-day aspirin, can be found in jasmine, beans, peas, clover and certain grasses and trees.

The ancient Egyptians used willow bark as a remedy for aches and pains, said Diarmuid Jeffreys, author of "Aspirin: The Remarkable Story of a Wonder Drug." They didn't know that what was reducing body temperature and inflammation was the salicylic acid.

Hippocrates, the Greek physician who lived from about 460 to 377 B.C., wrote that willow leaves and bark relieved pain and fevers.

It wasn't until thousands of years later that people began to isolate the key ingredients of aspirin. An 18th-century clergyman, Edward Stone, rediscovered aspirin, in effect, when he wrote a report about how a preparation of powdered willow bark seemed to benefit 50 patients with ague and other maladies, Roueché wrote.

In the 1800s, researchers across Europe explored salicylic acid. French pharmacist Henri Leroux isolated it in 1829, Roueché writes. Hermann Kolbe discovered synthetic salicylic acid in 1874, but when administered often in large doses, patients experienced nausea and vomiting, and some even went into a coma. A buffer was needed to ease the effects of this acid on the stomach.

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~


There's a reason that the platelets rush into creating blood clots when faced with the spike protein in a virus or injection etc but perhaps taking aspirin (or the powdered bark of the willow tree or just chewing on a supple willow branch... ....).... could help to flush out blood clots (or prevent excessive clots)

that would otherwise cause serious problems ....

(of course research and care would be needed regarding the dose.... )

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~


I had a quick look to see what had been written about Aspirin and Covid 19.... there's going to be lots more but this was a random article which includes discussion about aspirin and covid complications...

https://www.bhf.org.uk/informationsu...-complications

One of the most serious complications of coronavirus infection can be the risk of developing dangerous blood clots.

Covid-19 is linked to tiny clots inside the smallest blood vessels of the lungs, which are thought to affect how well the lungs can get oxygen into the body, and so could lead to symptoms such as breathlessness. And serious complications linked to blood clotting, such as deep vein thrombosis, pulmonary embolism or stroke, have been found in Covid-19 patients who need treatment in intensive care.

Luckily, we have many treatments to prevent or stop limit blood clotting – including ones which have been safely used for many years to help treat and prevent heart and circulatory diseases. Doctors and researchers are now looking at several different types of drugs that stop blood clots, in order to help people with Covid-19.

just wanted to share the aspirin stuff while it was fresh in my mind after reading the opening post....

[Apulu]

Posted by jaybee (here)
Thank you for the opening post info, Tintin..... very educational and interesting (this is the only post I've read in this thread so far although I've already seen the Stew Peters / Jane Ruby video...)

While I was going through it a thought came to me...... ASPIRIN.....

How come we never hear anything/much about aspirin now...? Well I haven't noticed anything...

Could aspirin be something important that is being quietly ignored because it is even more accessible and cheaper than hyroxychloroquine and ivermectin.........?


And even if 'they' got it took off the shelves the bark of the willow tree could be used and was where aspirin came from in the first place....

just a couple of random links bringing this subject together ie aspirin with the OP...

https://www.mayoclinic.org/diseases-...y/art-20046797

Aspirin interferes with your blood's clotting action. When you bleed, your blood's clotting cells, called platelets, build up at the site of your wound. The platelets help form a plug that seals the opening in your blood vessel to stop bleeding.

But this clotting can also happen within the vessels that supply your heart with blood. If your blood vessels are already narrowed from atherosclerosis — the buildup of fatty deposits in your arteries — a fatty deposit in your vessel lining can burst.

Then, a blood clot can quickly form and block the artery. This prevents blood flow to the heart and causes a heart attack. Aspirin therapy reduces the clumping action of platelets — possibly preventing a heart attack.

http://edition.cnn.com/2010/HEALTH/1...ory/index.html

The word "aspirin" wasn't a coincidence. It comes from Spiraea, a biological genus of shrubs that includes natural sources of the drug's key ingredient: salicylic acid. This acid, resembling what's in modern-day aspirin, can be found in jasmine, beans, peas, clover and certain grasses and trees.

The ancient Egyptians used willow bark as a remedy for aches and pains, said Diarmuid Jeffreys, author of "Aspirin: The Remarkable Story of a Wonder Drug." They didn't know that what was reducing body temperature and inflammation was the salicylic acid.

Hippocrates, the Greek physician who lived from about 460 to 377 B.C., wrote that willow leaves and bark relieved pain and fevers.

It wasn't until thousands of years later that people began to isolate the key ingredients of aspirin. An 18th-century clergyman, Edward Stone, rediscovered aspirin, in effect, when he wrote a report about how a preparation of powdered willow bark seemed to benefit 50 patients with ague and other maladies, Roueché wrote.

In the 1800s, researchers across Europe explored salicylic acid. French pharmacist Henri Leroux isolated it in 1829, Roueché writes. Hermann Kolbe discovered synthetic salicylic acid in 1874, but when administered often in large doses, patients experienced nausea and vomiting, and some even went into a coma. A buffer was needed to ease the effects of this acid on the stomach.

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~


There's a reason that the platelets rush into creating blood clots when faced with the spike protein in a virus or injection etc but perhaps taking aspirin (or the powdered bark of the willow tree or just chewing on a supple willow branch... ....).... could help to flush out blood clots (or prevent excessive clots)

that would otherwise cause serious problems ....

(of course research and care would be needed regarding the dose.... )

~~~~~~~~~~~~~~~~~~~~~~~~~~~~~


I had a quick look to see what had been written about Aspirin and Covid 19.... there's going to be lots more but this was a random article which includes discussion about aspirin and covid complications...

https://www.bhf.org.uk/informationsu...-complications

One of the most serious complications of coronavirus infection can be the risk of developing dangerous blood clots.

Covid-19 is linked to tiny clots inside the smallest blood vessels of the lungs, which are thought to affect how well the lungs can get oxygen into the body, and so could lead to symptoms such as breathlessness. And serious complications linked to blood clotting, such as deep vein thrombosis, pulmonary embolism or stroke, have been found in Covid-19 patients who need treatment in intensive care.

Luckily, we have many treatments to prevent or stop limit blood clotting – including ones which have been safely used for many years to help treat and prevent heart and circulatory diseases. Doctors and researchers are now looking at several different types of drugs that stop blood clots, in order to help people with Covid-19.

just wanted to share the aspirin stuff while it was fresh in my mind after reading the opening post....

Yeah I think you're on to something there jaybee - good thinking - I've heard Dr Peter McCullough talk about using aspirin and other blood thinners to prevent clotting in covid cases - it was in his long interview on Vimeo from May 2021. There's another one from this July which is also long but I haven't been able to find that one when I looked recently.

[Sue (Ayt)]

Posted by jaybee (here)
Thank you for the opening post info, Tintin..... very educational and interesting (this is the only post I've read in this thread so far although I've already seen the Stew Peters / Jane Ruby video...)

While I was going through it a thought came to me...... ASPIRIN.....

How come we never hear anything/much about aspirin now...? Well I haven't noticed anything...

Could aspirin be something important that is being quietly ignored because it is even more accessible and cheaper than hyroxychloroquine and ivermectin.........?


And even if 'they' got it took off the shelves the bark of the willow tree could be used and was where aspirin came from in the first place....

just a couple of random links bringing this subject together ie aspirin with the OP...

Thanks for mentioning this, Jaybee.
When my husband had a small stroke a week or so after a major surgery, he was rushed to the ER, and treated solely with 2 small plain aspirin tablets, which dissolved the clot that the surgery had released. Since then, I always carry some aspirin with me in case of strokes.

There is always that balance to keep in mind between too much and not enough clotting, of course. But some people with over-clotting problems are (or a least, used to be) treated with low-dose aspirin to keep the clotting in line. I have noticed though, that the medical system has been switched greatly to prescribing expensive (and often dangerous) blood-thinning prescription medications and lots of people are taking these. Unfortunately, they often are contraindicated for using with many natural supplements, which is sad to me when people on blood thinners simply cannot take many natural healing blood-thinners, as they could result in bleeds. I suspect there has been a demonizing campaign being waged for a while against good old-fashioned aspirin. It was such a staple at one time, for inflammatory symptoms.
"Take two aspirin and call me in the morning" was a standard joke.